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@ARTICLE{Preuss:276233,
      author       = {S. F. Preuss$^*$ and D. Grieshober$^*$ and H. Augustin$^*$},
      title        = {{S}ystemic {R}eprogramming of {E}ndothelial {C}ell
                      {S}ignaling in {M}etastasis and {C}achexia.},
      journal      = {Physiology},
      volume       = {38},
      number       = {4},
      issn         = {1548-9213},
      address      = {Stanford, Calif.},
      publisher    = {HighWire Press},
      reportid     = {DKFZ-2023-01042},
      pages        = {189–202},
      year         = {2023},
      note         = {#EA:A190#LA:A190#},
      abstract     = {Proliferating cancer cells secrete a multitude of factors
                      impacting metabolism, interorgan communication, and tumor
                      progression. The distribution of tumor-derived factors to
                      distant organs occurs via the circulation, which provides an
                      extensive reactive surface lined by endothelial cells.
                      Primary tumor-derived proteins impact cancer progression by
                      modulating endothelial cell activation at the
                      (pre-)metastatic niche, which affects tumor cell
                      dissemination as well as the outgrowth of seeded metastatic
                      cells into overt tumors. In addition, new insight indicates
                      that endothelial cell signaling contributes to metabolic
                      symptoms of cancer, including cancer-associated cachexia,
                      opening a new field of vascular metabolism research. This
                      review addresses how tumor-derived factors systemically
                      affect endothelial cell signaling and activation and impact
                      distant organs as well as tumor progression.},
      subtyp        = {Review Article},
      keywords     = {Humans / Endothelial Cells / Cachexia / Signal Transduction
                      / Neoplasm Proteins / angiocrine signaling (Other) /
                      cachexia (Other) / metastasis (Other) / systemic signaling
                      (Other) / vascular endothelium (Other) / Neoplasm Proteins
                      (NLM Chemicals)},
      cin          = {A190},
      ddc          = {610},
      cid          = {I:(DE-He78)A190-20160331},
      pnm          = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
      pid          = {G:(DE-HGF)POF4-311},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:37222464},
      doi          = {10.1152/physiol.00001.2023},
      url          = {https://inrepo02.dkfz.de/record/276233},
}