TY - JOUR
AU - Schmalz, Michael
AU - Liang, Xiao-Xuan
AU - Wieser, Ines
AU - Gruschel, Caroline
AU - Muskalla, Lukas
AU - Stöckl, Martin Thomas
AU - Nitschke, Roland
AU - Linz, Norbert
AU - Leitenstorfer, Alfred
AU - Vogel, Alfred
AU - Ferrando-May, Elisa
TI - Dissection of DNA damage and repair pathways in live cells by femtosecond laser microirradiation and free-electron modeling.
JO - Proceedings of the National Academy of Sciences of the United States of America
VL - 120
IS - 25
SN - 0027-8424
CY - Washington, DC
PB - National Acad. of Sciences
M1 - DKFZ-2023-01165
SP - e2220132120
PY - 2023
N1 - #LA:W650#
AB - Understanding and predicting the outcome of the interaction of light with DNA has a significant impact on the study of DNA repair and radiotherapy. We report on a combination of femtosecond pulsed laser microirradiation at different wavelengths, quantitative imaging, and numerical modeling that yields a comprehensive picture of photon-mediated and free-electron-mediated DNA damage pathways in live cells. Laser irradiation was performed under highly standardized conditions at four wavelengths between 515 nm and 1,030 nm, enabling to study two-photon photochemical and free-electron-mediated DNA damage in situ. We quantitatively assessed cyclobutane pyrimidine dimer (CPD) and γH2AX-specific immunofluorescence signals to calibrate the damage threshold dose at these wavelengths and performed a comparative analysis of the recruitment of DNA repair factors xeroderma pigmentosum complementation group C (XPC) and Nijmegen breakage syndrome 1 (Nbs1). Our results show that two-photon-induced photochemical CPD generation dominates at 515 nm, while electron-mediated damage dominates at wavelengths ≥620 nm. The recruitment analysis revealed a cross talk between nucleotide excision and homologous recombination DNA repair pathways at 515 nm. Numerical simulations predicted electron densities and electron energy spectra, which govern the yield functions of a variety of direct electron-mediated DNA damage pathways and of indirect damage by •OH radicals resulting from laser and electron interactions with water. Combining these data with information on free electron-DNA interactions gained in artificial systems, we provide a conceptual framework for the interpretation of the wavelength dependence of laser-induced DNA damage that may guide the selection of irradiation parameters in studies and applications that require the selective induction of DNA lesions.
KW - DNA strand breaks (Other)
KW - nonlinear photodamage (Other)
KW - oxidative DNA damage (Other)
KW - reductive DNA damage (Other)
KW - wavelength selectivity (Other)
LB - PUB:(DE-HGF)16
C6 - pmid:37307476
DO - DOI:10.1073/pnas.2220132120
UR - https://inrepo02.dkfz.de/record/276790
ER -