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@ARTICLE{ElMalki:277130,
author = {K. El Malki and P. Wehling and F. Alt and R. Sandhoff$^*$
and S. Zahnreich and A. Ustjanzew and C. Wilzius$^*$ and M.
A. Brockmann and A. Wingerter and A. Russo$^*$ and O. Beck
and C. Sommer and M. Ottenhausen and K. B. M. Frauenknecht
and C. Paret$^*$ and J. Faber$^*$},
title = {{G}lucosylceramide {S}ynthase {I}nhibitors {I}nduce
{C}eramide {A}ccumulation and {S}ensitize {H}3{K}27 {M}utant
{D}iffuse {M}idline {G}lioma to {I}rradiation.},
journal = {International journal of molecular sciences},
volume = {24},
number = {12},
issn = {1422-0067},
address = {Basel},
publisher = {Molecular Diversity Preservation International},
reportid = {DKFZ-2023-01292},
pages = {9905},
year = {2023},
note = {HI-TRON},
abstract = {H3K27M mutant (mut) diffuse midline glioma (DMG) is a
lethal cancer with no effective cure. The glycosphingolipids
(GSL) metabolism is altered in these tumors and could be
exploited to develop new therapies. We tested the effect of
the glucosylceramide synthase inhibitors (GSI) miglustat and
eliglustat on cell proliferation, alone or in combination
with temozolomide or ionizing radiation. Miglustat was
included in the therapy protocol of two pediatric patients.
The effect of H3.3K27 trimethylation on GSL composition was
analyzed in ependymoma. GSI reduced the expression of the
ganglioside GD2 in a concentration and time-dependent manner
and increased the expression of ceramide, ceramide
1-phosphate, sphingosine, and sphingomyelin but not of
sphingosine 1-phosphate. Miglustat significantly increased
the efficacy of irradiation. Treatment with miglustat
according to dose recommendations for patients with
Niemann-Pick disease was well tolerated with manageable
toxicities. One patient showed a mixed response. In
ependymoma, a high concentration of GD2 was found only in
the presence of the loss of H3.3K27 trimethylation. In
conclusion, treatment with miglustat and, in general,
targeting GSL metabolism may offer a new therapeutic
opportunity and can be administered in close proximity to
radiation therapy. Alterations in H3K27 could be useful to
identify patients with a deregulated GSL metabolism.},
keywords = {GD2 (Other) / H3K27 mutant diffuse midline glioma (Other) /
eliglustat (Other) / ependymoma (Other) / glycosphingolipids
(Other) / miglustat (Other) / trimethylation (Other)},
cin = {A411 / FM01},
ddc = {540},
cid = {I:(DE-He78)A411-20160331 / I:(DE-He78)FM01-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37373053},
doi = {10.3390/ijms24129905},
url = {https://inrepo02.dkfz.de/record/277130},
}