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@ARTICLE{Vuckovic:278405,
author = {N. Vuckovic$^*$ and K. Hoppe-Seyler$^*$ and A. Riemer$^*$},
title = {{C}haracterization of {D}o{T}c2 4510-{I}dentifying {HPV}16
{P}resence in a {C}ervical {C}arcinoma {C}ell {L}ine
{P}reviously {C}onsidered to {B}e {HPV}-{N}egative.},
journal = {Cancers},
volume = {15},
number = {15},
issn = {2072-6694},
address = {Basel},
publisher = {MDPI},
reportid = {DKFZ-2023-01639},
pages = {3810},
year = {2023},
note = {#EA:F130#LA:F130#},
abstract = {Cervical cancer is the fourth leading cause of cancer
deaths in women, with over 340,000 women dying from this
disease in 2020. Almost all cases have an underlying
persistent infection with an oncogenic high-risk type of
human papillomavirus (HPV), mainly HPV16. While cervical
squamous cell carcinoma is hardly ever HPV-negative, a small
subset of adenocarcinoma exhibits absence of HPV, even after
disproval of false-negative testing results due to low viral
load. This proportion is evident in many cervical cancer
studies and is reflected in the repertoire of model cell
lines commonly used in research. As the viral origin of
cervical cancer makes it a disease preventable and
potentially treatable by immunotherapeutic approaches, it is
the focus of many studies. For pertinent research, both a
broad set of HPV-infected cervical carcinoma models are
required, as well as stringent negative controls. A
ubiquitously used HPV-negative cervical adenocarcinoma cell
line is C-33A. Another cervical cancer cell line is
available for purchase from the American Type Culture
Collection (ATCC), namely DoTc2 4510, described to be
HPV-negative and thus as a model for a rare gynecological
malignancy. Here, we present findings proving that DoTc2
4510 is, in fact, an HPV16-positive cell line. This we
assessed using a highly sensitive nested multiplex PCR
protocol adapted for the identification of 12 carcinogenic
HPV types and a second PCR targeting the HPV16 oncogenes E6
and E7. Subsequently, the protein expression of E6 and E7
was examined, as well as the expression of their target
proteins p53, p21, and p16INK4a, to assess E6/E7
functionality. Finally, to attest to the survival dependence
of DoTc2 4510 cells on HPV16, we performed an HPV16
E6/E7-targeted siRNA knock-down, which indeed led to
senescence induction. Together, these findings demonstrate
that DoTc2 4510 is an HPV16-transformed cell line.},
keywords = {DoTc2 4510 (Other) / HPV (Other) / HPV-negative cervical
cancer (Other) / cell authentication (Other) / cervical
cancer (Other)},
cin = {F130 / F065},
ddc = {610},
cid = {I:(DE-He78)F130-20160331 / I:(DE-He78)F065-20160331},
pnm = {316 - Infektionen, Entzündung und Krebs (POF4-316)},
pid = {G:(DE-HGF)POF4-316},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37568626},
pmc = {pmc:PMC10417116},
doi = {10.3390/cancers15153810},
url = {https://inrepo02.dkfz.de/record/278405},
}