TY  - JOUR
AU  - Briest, Franziska
AU  - Noerenberg, Daniel
AU  - Hennch, Cornelius
AU  - Yoshida, Kenichi
AU  - Hablesreiter, Raphael
AU  - Nimo, Jose
AU  - Sasca, Daniel
AU  - Kirchner, Marieluise
AU  - Mansouri, Larry
AU  - Inoue, Yoshikage
AU  - Wiegand, Laura
AU  - Staiger, Annette M
AU  - Casadei, Beatrice
AU  - Korkolopoulou, Penelope
AU  - Weiner, January
AU  - Lopez-Guillermo, Armando
AU  - Warth, Arne
AU  - Schneider, Tamás
AU  - Nagy, Ákos
AU  - Klapper, Wolfram
AU  - Hummel, Michael
AU  - Kanellis, George
AU  - Anagnostopoulos, Ioannis
AU  - Mertins, Philipp
AU  - Bullinger, Lars
AU  - Rosenquist, Richard
AU  - Vassilakopoulos, Theodoros P
AU  - Ott, German
AU  - Ogawa, Seishi
AU  - Damm, Frederik
TI  - Frequent ZNF217 mutations lead to transcriptional deregulation of interferon signal transduction via altered chromatin accessibility in B cell lymphoma.
JO  - Leukemia
VL  - 37
IS  - 11
SN  - 0887-6924
CY  - London
PB  - Springer Nature
M1  - DKFZ-2023-01765
SP  - 2237-2249
PY  - 2023
N1  - 2023 Nov;37(11):2237-2249
AB  - Recent exome-wide studies discovered frequent somatic mutations in the epigenetic modifier ZNF217 in primary mediastinal B cell lymphoma (PMBCL) and related disorders. As functional consequences of ZNF217 alterations remain unknown, we comprehensively evaluated their impact in PMBCL. Targeted sequencing identified genetic lesions affecting ZNF217 in 33
LB  - PUB:(DE-HGF)16
C6  - pmid:37648814
DO  - DOI:10.1038/s41375-023-02013-9
UR  - https://inrepo02.dkfz.de/record/282445
ER  -