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@ARTICLE{Heidegger:282500,
author = {S. Heidegger and F. Stritzke and S. Dahl and J.
Daßler-Plenker and L. Joachim and D. Buschmann and K. Fan
and C. M. Sauer and N. Ludwig and C. Winter$^*$ and S.
Enssle and S. Li and M. Perl and A. Görgens and T. Haas and
E. T. Orberg$^*$ and S. Göttert and C. Wölfel$^*$ and T.
Engleitner and I. Cortés-Ciriano and R. Rad$^*$ and W. Herr
and B. Giebel and J. Ruland$^*$ and F. Bassermann$^*$ and C.
Coch and G. Hartmann and H. Poeck},
title = {{T}argeting nucleic acid sensors in tumor cells to
reprogram biogenesis and {RNA} cargo of extracellular
vesicles for {T} cell-mediated cancer immunotherapy.},
journal = {Cell reports / Medicine},
volume = {4},
number = {9},
issn = {2666-3791},
address = {Maryland Heights, MO},
publisher = {Elsevier},
reportid = {DKFZ-2023-01789},
pages = {101171},
year = {2023},
note = {2023 Sep 19;4(9):101171},
abstract = {Tumor-derived extracellular vesicles (EVs) have been
associated with immune evasion and tumor progression. We
show that the RNA-sensing receptor RIG-I within tumor cells
governs biogenesis and immunomodulatory function of EVs.
Cancer-intrinsic RIG-I activation releases EVs, which
mediate dendritic cell maturation and T cell antitumor
immunity, synergizing with immune checkpoint blockade.
Intact RIG-I, autocrine interferon signaling, and the GTPase
Rab27a in tumor cells are required for biogenesis of
immunostimulatory EVs. Active intrinsic RIG-I signaling
governs composition of the tumor EV RNA cargo including
small non-coding stimulatory RNAs. High transcriptional
activity of EV pathway genes and RIG-I in melanoma samples
associate with prolonged patient survival and beneficial
response to immunotherapy. EVs generated from human melanoma
after RIG-I stimulation induce potent antigen-specific T
cell responses. We thus define a molecular pathway that can
be targeted in tumors to favorably alter EV immunomodulatory
function. We propose 'reprogramming' of tumor EVs as a
personalized strategy for T cell-mediated cancer
immunotherapy.},
keywords = {RIG-I (Other) / RNA (Other) / STING (Other) / cancer
immunotherapy (Other) / cancer resistance (Other) /
extracellular vesicles (Other) / innate immunity (Other) /
nucleic acid receptors (Other) / personalized therapy
(Other)},
cin = {MU01 / FM01},
ddc = {610},
cid = {I:(DE-He78)MU01-20160331 / I:(DE-He78)FM01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37657445},
doi = {10.1016/j.xcrm.2023.101171},
url = {https://inrepo02.dkfz.de/record/282500},
}