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@ARTICLE{Karur:283172,
author = {G. R. Karur and A. Aneja and J. Stojanovska and K. Hanneman
and R. Latchamsetty and D. Kersting$^*$ and P. S. Rajiah},
title = {{I}maging of {C}ardiac {F}ibrosis: {A}n {U}pdate, {F}rom
the {AJR} {S}pecial {S}eries on {I}maging of {F}ibrosis.},
journal = {American journal of roentgenology},
volume = {222},
number = {6},
issn = {0002-9580},
address = {Stanford, Calif.},
publisher = {Highwire Press},
reportid = {DKFZ-2023-01951},
pages = {e2329870},
year = {2024},
note = {2024 Jun;222(6):e2329870},
abstract = {Myocardial fibrosis (MF) is defined as excessive production
and deposition of extracellular matrix (ECM) proteins,
resulting in pathologic myocardial remodeling. Three types
of MF have been identified: replacement fibrosis from tissue
necrosis, reactive fibrosis from myocardial stress, and
infiltrative interstitial fibrosis from progressive
deposition of non-degradable material such as amyloid. While
echocardiography, nuclear medicine, and CT play important
roles in the assessment of MF, MRI is pivotal in the
evaluation of MF, using the late gadolinium enhancement
(LGE) technique as a primary endpoint. The LGE technique
focuses on the pattern and distribution of gadolinium
accumulation in the myocardium and assists the diagnosis and
establishment of the etiology of both ischemic and
non-ischemic cardiomyopathy. LGE MRI aids prognostication
and risk stratification. In addition, LGE MRI is used to
guide management of patients being considered for ablation
for arrhythmias. Parametric mapping techniques, including T1
mapping and extracellular volume measurement, allow
detection and quantification of diffuse fibrosis, which may
not be detected by LGE MRI. These techniques also allow
monitoring of disease progression and therapy response. This
review provides an update on imaging of MF, including
prognostication and risk stratification tools,
electrophysiologic considerations, and disease monitoring.},
subtyp = {Review Article},
cin = {ED01},
ddc = {610},
cid = {I:(DE-He78)ED01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:37753860},
doi = {10.2214/AJR.23.29870},
url = {https://inrepo02.dkfz.de/record/283172},
}