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@ARTICLE{Duran:287620,
author = {I. Duran and J. Pombo and B. Sun and S. U. Gallage$^*$ and
H. Kudo and D. McHugh and L. Bousset and J. E. Barragan
Avila$^*$ and R. Forlano and P. Manousou and M.
Heikenwalder$^*$ and D. J. Withers and S. Vernia and R. D.
Goldin and J. Gil},
title = {{D}etection of senescence using machine learning algorithms
based on nuclear features.},
journal = {Nature Communications},
volume = {15},
number = {1},
issn = {2041-1723},
address = {[London]},
publisher = {Nature Publishing Group UK},
reportid = {DKFZ-2024-00274},
pages = {1041},
year = {2024},
abstract = {Cellular senescence is a stress response with broad
pathophysiological implications. Senotherapies can induce
senescence to treat cancer or eliminate senescent cells to
ameliorate ageing and age-related pathologies. However, the
success of senotherapies is limited by the lack of reliable
ways to identify senescence. Here, we use nuclear morphology
features of senescent cells to devise machine-learning
classifiers that accurately predict senescence induced by
diverse stressors in different cell types and tissues. As a
proof-of-principle, we use these senescence classifiers to
characterise senolytics and to screen for drugs that
selectively induce senescence in cancer cells but not normal
cells. Moreover, a tissue senescence score served to assess
the efficacy of senolytic drugs and identified senescence in
mouse models of liver cancer initiation, ageing, and
fibrosis, and in patients with fatty liver disease. Thus,
senescence classifiers can help to detect pathophysiological
senescence and to discover and validate potential
senotherapies.},
cin = {F180 / D440},
ddc = {500},
cid = {I:(DE-He78)F180-20160331 / I:(DE-He78)D440-20160331},
pnm = {314 - Immunologie und Krebs (POF4-314)},
pid = {G:(DE-HGF)POF4-314},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:38310113},
doi = {10.1038/s41467-024-45421-w},
url = {https://inrepo02.dkfz.de/record/287620},
}