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@ARTICLE{Sha:289179,
      author       = {S. Sha$^*$ and L.-J. Chen$^*$ and H. Brenner$^*$ and B.
                      Schöttker$^*$},
      title        = {{S}erum 25-{H}ydroxyvitamin {D} {S}tatus and {V}itamin {D}
                      {S}upplements {U}se {A}re {N}ot {A}ssociated with {L}ow
                      {B}ack {P}ain in the {L}arge {UK} {B}iobank {C}ohort.},
      journal      = {Nutrients},
      volume       = {16},
      number       = {6},
      issn         = {2072-6643},
      address      = {Basel},
      publisher    = {MDPI},
      reportid     = {DKFZ-2024-00614},
      pages        = {806},
      year         = {2024},
      note         = {#EA:C070#LA:C070#},
      abstract     = {Longitudinal studies assessing the association of vitamin D
                      deficiency, defined by serum 25-hydroxyvitamin D levels < 30
                      nmol/L, and vitamin D supplement (VDS) use with low back
                      pain (LBP) are sparse. This investigation assessed the
                      cross-sectional and longitudinal association of vitamin D
                      status and VDS use with LBP among 135,934 participants from
                      the UK Biobank cohort. Approximately $21.6\%$ of the
                      participants had vitamin D deficiency, while only $4\%$
                      regularly took VDS. In the month before study enrollment,
                      $3.8\%$ of the population reported experiencing LBP. An
                      additional $3.3\%$ of the population were diagnosed with LBP
                      by their general practitioners for the first time during a
                      median follow-up of 8.5 years. Vitamin D deficiency and VDS
                      use were cross-sectionally associated with LBP in age- and
                      sex-adjusted models, but these associations were not evident
                      in comprehensively adjusted models. In longitudinal
                      analyses, both vitamin D deficiency and VDS use were not
                      associated with LBP in any model after correction for
                      multiple testing. In conclusion, not unexpectedly due to the
                      fact that LBP is multifactorial, our findings provide no
                      evidence for the role of the vitamin D status in the
                      etiology of LBP.},
      keywords     = {25-hydroxyvitamin D (Other) / low back pain (Other) /
                      real-world evidence (Other) / vitamin D supplement use
                      (Other)},
      cin          = {C070 / C120},
      ddc          = {610},
      cid          = {I:(DE-He78)C070-20160331 / I:(DE-He78)C120-20160331},
      pnm          = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
      pid          = {G:(DE-HGF)POF4-313},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:38542718},
      doi          = {10.3390/nu16060806},
      url          = {https://inrepo02.dkfz.de/record/289179},
}