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@ARTICLE{Stocker:291769,
author = {H. Stocker$^*$ and M. Gentiluomo and K. Trares$^*$ and L.
Beyer and J. Stevenson-Hoare$^*$ and D. Rujescu and B.
Holleczek and K. Beyreuther and K. Gerwert and B.
Schöttker$^*$ and D. Campa and F. Canzian$^*$ and H.
Brenner$^*$},
title = {{M}itochondrial {DNA} abundance in blood is associated with
{A}lzheimer's disease- and dementia-risk.},
journal = {Molecular psychiatry},
volume = {30},
issn = {1359-4184},
address = {London},
publisher = {Macmillan},
reportid = {DKFZ-2024-01505},
pages = {131–139},
year = {2025},
note = {#EA:C070#LA:C070# / 30, pages 131–139 (2025)},
abstract = {The mitochondrial cascade hypothesis of Alzheimer's disease
(AD) has been portrayed through molecular, cellular, and
animal studies; however large epidemiological studies are
lacking. This study aimed to explore the association of
mitochondrial DNA copy number (mtDNAcn), a marker
representative of mtDNA abundance per cell, with risk of
incident all-cause dementia, AD, and vascular dementia
diagnosis within 17 years and dementia-related blood
biomarkers (P-tau181, GFAP, and NfL). Additionally,
sex-stratified analyses were completed. In this German
population-based cohort study (ESTHER), 9940 participants
aged 50-75 years were enrolled by general practitioners and
followed for 17 years. Participants were included in this
study if information on dementia status and blood-based
mtDNAcn measured via real-time polymerase chain reaction
were available. In a nested case-control approach, a
subsample of participants additionally had measurements of
P-tau181, GFAP, and NfL in blood samples taken at baseline.
Of 4913 participants eligible for analyses, 386 were
diagnosed with incident all-cause dementia, including 130 AD
and 143 vascular dementia cases, while 4527 participants
remained without dementia diagnosis within 17 years.
Participants with low mtDNAcn (lowest $10\%)$ experienced
$45\%$ and $65\%$ percent increased risk of incident
all-cause dementia and AD after adjusting for age and sex
(all-cause dementia: HRadj, $95\%CI:1.45,$ 1.08-1.94; AD:
HRadj, $95\%CI:$ 1.65, 1.01-2.68). MtDNAcn was not
associated to vascular dementia diagnosis and was more
strongly associated with all-cause dementia among women. In
the nested case-control study (n = 790), mtDNAcn was not
significantly associated with the dementia-related blood
biomarkers (P-tau181, GFAP, and NfL) levels in blood from
baseline before dementia diagnosis. This study provides
novel epidemiological evidence connecting mtDNA abundance,
measured via mtDNAcn, to incident dementia and AD at the
population-based level. Reduced mitochondrial abundance may
play a role in pathogenesis, especially among women.},
cin = {C070 / C055},
ddc = {610},
cid = {I:(DE-He78)C070-20160331 / I:(DE-He78)C055-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39009700},
doi = {10.1038/s41380-024-02670-x},
url = {https://inrepo02.dkfz.de/record/291769},
}