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@ARTICLE{Huybrechts:292355,
author = {I. Huybrechts and B. Chimera and G. T. Hanley-Cook and C.
Biessy and M. Deschasaux-Tanguy and M. Touvier and E.
Kesse-Guyot and B. Srour and J. Baudry and J. Berlivet and
C. Casagrande and G. Nicolas and J. B. Lopez and C. J.
Millett and E. K. Cakmak and O. J. K. Robinson and K. A.
Murray and M. B. Schulze and G. Masala and M. Guevara and S.
Bodén and A. J. Cross and K. Tsilidis and A. K. Heath and
S. Panico and P. Amiano and J. M. Huerta and T. Key and U.
Ericson and T. Stocks and M. W. Lundblad and G. Skeie and C.
Sacerdote and V. Katzke$^*$ and M. C. Playdon and P. Ferrari
and P. Vineis and C. Lachat and M. J. Gunter},
title = {{F}ood biodiversity and gastrointestinal cancer risk in
nine {E}uropean countries: {A}nalysis within a prospective
cohort study.},
journal = {European journal of cancer},
volume = {210},
issn = {0014-2964},
address = {Amsterdam [u.a.]},
publisher = {Elsevier},
reportid = {DKFZ-2024-01704},
pages = {114258},
year = {2024},
abstract = {Food biodiversity in human diets has potential co-benefits
for both public health and sustainable food systems.
However, current evidence on the potential relationship
between food biodiversity and cancer risk, and particularly
gastrointestinal cancers typically related to diet, remains
limited. This study evaluated how dietary species richness
(DSR) was associated with gastrointestinal cancer risk in a
pan-European population.Associations between DSR and
subsequent gastrointestinal cancer risk were examined among
450,111 adults enrolled in the European Prospective
Investigation into Cancer and Nutrition cohort (EPIC,
initiated in 1992), free of cancer at baseline. Usual
dietary intakes were assessed at recruitment with
country-specific dietary questionnaires. DSR of an
individual's yearly diet was calculated based on the
absolute number of unique biological species in each food
and drink item. Associations between DSR and cancer risk
were assessed by multivariable Cox proportional hazards
regression models.During a median follow-up time of 14.1
years (SD=3.9), 10,705 participants were diagnosed with
gastrointestinal cancer. Hazard ratios (HRs) and 95 $\%$
confidence intervals (CIs) comparing overall
gastrointestinal cancer risk in the highest versus lowest
quintiles of DSR indicated inverse associations in
multivariable-adjusted models [HR (95 $\%$ CI): 0.77
(0.69-0.87); P-value < 0·0001] (Table 2). Specifically,
inverse associations were observed between DSR and
oesophageal squamous cell carcinoma, proximal colon,
colorectal, and liver cancer risk (p-trend<0.05 for all
cancer types).Greater food biodiversity in the diet may
lower the risk of certain gastrointestinal cancers. Further
research is needed to replicate these novel findings and to
understand potential mechanisms.},
cin = {C020},
ddc = {610},
cid = {I:(DE-He78)C020-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39168001},
doi = {10.1016/j.ejca.2024.114258},
url = {https://inrepo02.dkfz.de/record/292355},
}