% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Damerell:293587,
author = {V. Damerell and N. Klaassen-Dekker and S. Brezina and J.
Ose and A. Ulvik and E. H. van Roekel and A. N. Holowatyj
and A. Baierl and J. Böhm and M. J. L. Bours and H.
Brenner$^*$ and J. H. W. de Wilt and W. M. Grady and N.
Habermann and M. Hoffmeister$^*$ and P. Keski-Rahkonen and
T. Lin and P. Schirmacher and P. Schrotz-King$^*$ and A. B.
Ulrich and F. J. B. van Duijnhoven and C. A. Warby and D.
Shibata and A. T. Toriola and J. C. Figueiredo and E. M.
Siegel and C. I. Li and A. Gsur and E. Kampman and M.
Schneider and P. M. Ueland and M. P. Weijenberg and C. M.
Ulrich and D. E. Kok and B. Gigic},
collaboration = {F. Consortium},
title = {{C}irculating tryptophan-kynurenine pathway metabolites are
associated with all-cause mortality among patients with
stage {I}-{III} colorectal cancer.},
journal = {International journal of cancer},
volume = {156},
number = {3},
issn = {0020-7136},
address = {Bognor Regis},
publisher = {Wiley-Liss},
reportid = {DKFZ-2024-01910},
pages = {552-565},
year = {2025},
note = {2025 Feb 1;156(3):552-565},
abstract = {Alterations within the tryptophan-kynurenine metabolic
pathway have been linked to the etiology of colorectal
cancer (CRC), but the relevance of this pathway for
prognostic outcomes in CRC patients needs further
elucidation. Therefore, we investigated associations between
circulating concentrations of tryptophan-kynurenine pathway
metabolites and all-cause mortality among CRC patients. This
study utilizes data from 2102 stage I-III CRC patients
participating in six prospective cohorts involved in the
international FOCUS Consortium. Preoperative circulating
concentrations of tryptophan, kynurenine, kynurenic acid
(KA), 3-hydroxykynurenine (HK), xanthurenic acid (XA),
3-hydroxyanthranilic acid (HAA), anthranilic acid (AA),
picolinic acid (PA), and quinolinic acid (QA) were measured
by liquid chromatography-tandem mass spectrometry. Using Cox
proportional hazards regression, we examined associations of
above-mentioned metabolites with all-cause mortality,
adjusted for potential confounders. During a median
follow-up of 3.2 years (interquartile range: 2.2-4.9), 290
patients $(13.8\%)$ deceased. Higher blood concentrations of
tryptophan, XA, and PA were associated with a lower risk of
all-cause mortality (per doubling in concentrations:
tryptophan: HR = 0.56; $95\%CI:0.41,0.76,$ XA: HR = 0.74;
$95\%CI:0.64,0.85,$ PA: HR = 0.76; $95\%CI:0.64,0.92),$
while higher concentrations of HK and QA were associated
with an increased risk of death (per doubling in
concentrations: HK: HR = 1.80; $95\%CI:1.47,2.21,$ QA: HR =
1.31; $95\%CI:1.05,1.63).$ A higher kynurenine-to-tryptophan
ratio, a marker of cell-mediated immune activation, was
associated with an increased risk of death (per doubling: HR
= 2.07; $95\%CI:1.52,2.83).$ In conclusion,
tryptophan-kynurenine pathway metabolites may be prognostic
markers of survival in CRC patients.},
keywords = {all‐cause mortality (Other) / colorectal cancer (Other) /
kynurenines (Other) / prognosis (Other) / tryptophan
(Other)},
cin = {C070 / C120 / HD01},
ddc = {610},
cid = {I:(DE-He78)C070-20160331 / I:(DE-He78)C120-20160331 /
I:(DE-He78)HD01-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39308420},
doi = {10.1002/ijc.35183},
url = {https://inrepo02.dkfz.de/record/293587},
}