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@ARTICLE{SimovicLorenz:294547,
author = {M. Simovic-Lorenz$^*$ and A. Ernst$^*$},
title = {{C}hromothripsis in cancer.},
journal = {Nature reviews / Cancer},
volume = {25},
number = {2},
issn = {1474-175X},
address = {London [u.a.]},
publisher = {Nature Publ. Group},
reportid = {DKFZ-2024-02322},
pages = {79–92},
year = {2025},
note = {#EA:B420#LA:B420# / Volume 25 | February 2025 | 79–92},
abstract = {Chromothripsis is a mutational phenomenon in which a single
catastrophic event generates extensive rearrangements of one
or a few chromosomes. This extreme form of genome
instability has been detected in $30-50\%$ of cancers.
Studies conducted in the past few years have uncovered
insights into how chromothripsis arises and deciphered some
of the cellular and molecular consequences of chromosome
shattering. This Review discusses the defining features of
chromothripsis and describes its prevalence across different
cancer types as indicated by the manifestations of
chromothripsis detected in human cancer samples. The
different mechanistic models of chromothripsis, derived from
in vitro systems that enable causal inference through
experimental manipulation, are discussed in detail. The
contribution of chromothripsis to cancer development, the
selective advantages that cancer cells might gain from
chromothripsis, the evolutionary trajectories of
chromothriptic tumours, and the potential vulnerabilities
and therapeutic opportunities presented by chromothriptic
cells are also highlighted.},
subtyp = {Review Article},
cin = {B420 / HD01},
ddc = {610},
cid = {I:(DE-He78)B420-20160331 / I:(DE-He78)HD01-20160331},
pnm = {312 - Funktionelle und strukturelle Genomforschung
(POF4-312)},
pid = {G:(DE-HGF)POF4-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39548283},
doi = {10.1038/s41568-024-00769-5},
url = {https://inrepo02.dkfz.de/record/294547},
}