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@ARTICLE{KarimianJazi:294843,
author = {K. Karimian-Jazi and N. Enbergs and E. Golubtsov and K.
Schregel and J. Ungermann and H. Fels-Palesandro and D.
Schwarz and V. Sturm and J. M. Kernbach and D. Batra and F.
Ippen$^*$ and I. Pflüger and N. von Knebel Doeberitz$^*$
and S. Heiland and L. Bunse$^*$ and M. Platten$^*$ and F.
Winkler$^*$ and W. Wick$^*$ and D. Paech$^*$ and M. Bendszus
and M. Breckwoldt$^*$},
title = {{D}ifferentiating {G}lioma {R}ecurrence and
{P}seudoprogression by {APT}w {CEST} {MRI}.},
journal = {Investigative radiology},
volume = {60},
number = {6},
issn = {0020-9996},
address = {[Erscheinungsort nicht ermittelbar]},
publisher = {Ovid},
reportid = {DKFZ-2024-02553},
pages = {414-422},
year = {2025},
note = {#EA:B320#LA:D170# / 2025 Jun 1;60(6):414-422},
abstract = {Recurrent glioma is highly treatment resistant due to its
metabolic, cellular, and molecular heterogeneity and
invasiveness. Tumor monitoring by conventional MRI has
shortcomings to assess these key glioma characteristics.
Recent studies introduced chemical exchange saturation
transfer for metabolic imaging in oncology and assessed its
diagnostic value for newly diagnosed glioma. This
prospective study investigates amide proton
transfer-weighted (APTw) MRI at 3 T as an imaging biomarker
to elucidate the molecular heterogeneity and invasion
patterns of recurrent glioma in comparison to
pseudoprogression (PsPD).We performed a monocenter,
prospective trial and screened 371 glioma patients who
received tumor monitoring between August 2021 and March 2024
at our institution. The study included IDH wildtype
astrocytoma and IDH mutant astrocytoma and
oligodendroglioma, graded according to the WHO 2021
classification. Patients had received clinical standard of
care treatment including surgical resection and
radiochemotherapy prior to study inclusion. Patients were
monitored by 3 monthly MRI follow-up imaging, and response
assessment was performed according to the RANO criteria.
Within this cohort, we identified 30 patients who presented
with recurrent glioma and 12 patients with PsPD. In addition
to standard anatomical sequences (FLAIR and T1-w Gd-enhanced
sequences), MRI included APTw imaging. After sequence
co-registration, semiautomated segmentation was performed of
the FLAIR lesion, CE lesion, resection cavity, and the
contralateral normal-appearing white matter, and APTw
signals were quantified in these regions of interest.APTw
values were highest in solid, Gd-enhancing tumor parts as
compared with the nonenhancing FLAIR lesion (APTw: $1.99\%$
vs $1.36\%,$ P = 0.001), whereas there were no detectable
APTw alterations in the normal-appearing white matter (APTw:
$0.005\%,$ P < 0.001 compared with FLAIR). Patients with
progressive disease had higher APTw levels compared with
patients with PsPD (APTw: $1.99\%$ vs $1.26\%,$ P = 0.008).
Chemical exchange saturation transfer identified
heterogeneity within the FLAIR lesion that was not
detectable by conventional sequences. There were also focal
APTw signal peaks within contrast enhancing lesions as
putative metabolic hotspots within recurrent glioma. The
resection cavity developed an APTw increase at recurrence
that was not detectable prior to recurrence nor in patients
with PsPD (APTw before recurrence: $0.6\%$ vs $2.68\%$ at
recurrence, P = 0.03).Our study shows that APTw imaging can
differentiate PD and PsPD. We identify previously
undetectable imaging patterns during glioma recurrence,
which include alterations within resection cavity associated
with disease progression. Our work highlights the clinical
potential of APTw imaging for glioma monitoring and further
establishes it as an imaging biomarker in neuro-oncology.},
cin = {B320 / HD01 / B300 / E010 / D170},
ddc = {610},
cid = {I:(DE-He78)B320-20160331 / I:(DE-He78)HD01-20160331 /
I:(DE-He78)B300-20160331 / I:(DE-He78)E010-20160331 /
I:(DE-He78)D170-20160331},
pnm = {314 - Immunologie und Krebs (POF4-314)},
pid = {G:(DE-HGF)POF4-314},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39644107},
doi = {10.1097/RLI.0000000000001145},
url = {https://inrepo02.dkfz.de/record/294843},
}