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@ARTICLE{Logotheti:299831,
author = {S. Logotheti$^*$ and S. N. Vasilopoulos and I. Tremi and A.
Gkikoudi and M. Fragkos and A. Pavlopoulou and S. Havaki and
A. G. Georgakilas},
title = {{T}he {DNA} {D}amage {R}esponse as an {A}uxiliary
{I}ndicator of {S}enescence in {C}ancer: {A}
{U}ser-{F}riendly {T}oolkit of {M}arkers and {D}etection
{M}ethods.},
journal = {Methods in molecular biology},
volume = {2906},
issn = {1064-3745},
address = {Totowa, NJ},
publisher = {Humana Press},
reportid = {DKFZ-2025-00568},
isbn = {978-1-0716-4425-6 (print)},
pages = {83-112},
year = {2025},
note = {#EA:E041#},
abstract = {Senescence is a cellular stress response causing a stable
exit from the cell cycle. Limitation of cell proliferation
is accompanied by a variety of characteristic changes,
including cellular and nuclear morphological alterations,
chromatin rearrangements, metabolic reprogramming, nuclear
envelope rupture, and an immunomodulatory secretome, termed
senescence-associated secreted phenotype (SASP). Senescence
is a robust mechanism of tissue homeostasis, with crucial
roles in eliminating dysfunctional and/or premalignant
cells, and tissue repair. Activation of oncogenes triggers
senescence, which in turn arrests DNA-damaged cells. This
type of response, known as oncogene-induced senescence (OIS)
is a powerful barrier to oncogenesis. However, in specific
contexts, senescent cells support tumor progression, mainly
via their secretome-mediated activities. The dynamic,
context-dependent and ambivalent nature of senescence
challenges the identification, characterization, and
specific targeting of senescent cells for disease
management. Importantly, senescence is highly intertwined
with DNA damage response and repair (DDR/R) machinery that
safeguards genome integrity. DNA damage events are
frequently prevalent, while DDR/R signaling pathways are
active during cellular senescence. In this regard, it can
serve as an auxiliary indicator for senescent cells. In this
chapter, we emphasize DDR/R as a shared hallmark of cancer
and senescence. We provide a wieldy toolkit of experimental
methods for monitoring DDR-related senescence markers, and
we demonstrate the potential of using natural language
processing to extract additional DDR/R biomarkers tailored
to specific cancer types. Our methodologies can facilitate a
comprehensive study of senescence in aging and cancer.},
keywords = {Humans / Cellular Senescence: genetics / DNA Damage /
Neoplasms: genetics / Neoplasms: metabolism / Neoplasms:
pathology / DNA Repair / Senescence-Associated Secretory
Phenotype / Biomarkers, Tumor: metabolism / Biomarkers,
Tumor: genetics / Cell Line, Tumor / Biomarkers / Biomarkers
(Other) / DNA Damage Response (DDR) (Other) / DNA damage
(Other) / Damage Response and Repair (DDR/R) (Other) /
Microscopy (Other) / Senescence (Other) / Biomarkers, Tumor
(NLM Chemicals) / Biomarkers (NLM Chemicals)},
cin = {E041},
ddc = {570},
cid = {I:(DE-He78)E041-20160331},
pnm = {315 - Bildgebung und Radioonkologie (POF4-315)},
pid = {G:(DE-HGF)POF4-315},
typ = {PUB:(DE-HGF)3 / PUB:(DE-HGF)16},
pubmed = {pmid:40082352},
doi = {DOI:10.1007/978-1-0716-4426-3_6},
url = {https://inrepo02.dkfz.de/record/299831},
}