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@ARTICLE{Gutu:300146,
author = {N. Gutu and H. Ishikuma and C. Ector and U. Keilholz and H.
Herzel and A. E. Granada$^*$},
title = {{A} combined mathematical and experimental approach reveals
the drivers of time-of-day drug sensitivity in human cells.},
journal = {Communications biology},
volume = {8},
number = {1},
issn = {2399-3642},
address = {London},
publisher = {Springer Nature},
reportid = {DKFZ-2025-00637},
pages = {491},
year = {2025},
abstract = {The circadian clock plays a pivotal role in regulating
various aspects of cancer, influencing tumor growth and
treatment responses. There are significant changes in drug
efficacy and adverse effects when drugs are administered at
different times of the day, underscoring the importance of
considering the time of day in treatments. Despite these
well-established findings, chronotherapy approaches in drug
treatment have yet to fully integrate into clinical
practice, largely due to the stringent clinical requirements
for proving efficacy and safety, alongside the need for
deeper mechanistic insights. In this study, we employ a
combined mathematical and experimental approach to
systematically investigate the factors influencing
time-of-day drug sensitivity in human cells. Here we show
how circadian and drug properties independently shape
time-of-day profiles, providing valuable insights into the
temporal dynamics of treatment responses. Understanding how
drug efficacy fluctuates throughout the day holds immense
potential for the development of personalized treatment
strategies aligned with an individual's internal biological
clock, revolutionizing cancer treatment by maximizing
therapeutic benefits. Moreover, our framework offers a
promising avenue for refining future drug screening efforts,
paving the way for more effective and targeted therapies
across diverse tissue types.},
keywords = {Humans / Circadian Clocks: drug effects / Antineoplastic
Agents: pharmacology / Neoplasms: drug therapy / Neoplasms:
pathology / Circadian Rhythm: drug effects / Cell Line,
Tumor / Models, Biological / Drug Resistance, Neoplasm /
Antineoplastic Agents (NLM Chemicals)},
cin = {BE01},
ddc = {570},
cid = {I:(DE-He78)BE01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40133704},
doi = {10.1038/s42003-025-07931-1},
url = {https://inrepo02.dkfz.de/record/300146},
}