% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Schoeps:300181,
author = {B. Schoeps and U. M. Lauer$^*$ and K. Elbers},
title = {{D}eciphering permissivity of human tumor ecosystems to
oncolytic viruses.},
journal = {Oncogene},
volume = {44},
number = {16},
issn = {0950-9232},
address = {London},
publisher = {Springer Nature},
reportid = {DKFZ-2025-00658},
pages = {1069-1077},
year = {2025},
note = {2025 May;44(16):1069-1077},
abstract = {Effective cancer therapy involves initiation of a
tumor-specific immune response. Consequently, the interest
in oncolytic viruses (OV) capable of triggering immunogenic
cell death has sparked in recent years. However, the common
use of pre-clinical models that fail to mirror patient tumor
ecosystems (TES) hinders clinical translation. Here, we
provide a condensed view on the intricate interplay between
several aspects of TES and OV action and discuss these
considerations in the view of recently developed
pre-clinical human model systems. Given the urgent demand
for innovative cancer treatments, the purpose of this review
is to highlight the so-far overlooked complex impact of the
tumor microenvironment (TME) on OV permissivity, with the
intent to provide a foundation for future, more effective
pre-clinical studies.},
subtyp = {Review Article},
cin = {TU01},
ddc = {610},
cid = {I:(DE-He78)TU01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40148688},
doi = {10.1038/s41388-025-03357-5},
url = {https://inrepo02.dkfz.de/record/300181},
}