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@ARTICLE{VanBergen:300233,
author = {T. D. Van Bergen and A. J. A. T. Braat and R. Hermsen and
J. G. Heetman and L. Wever and J. Lavalaye and M. Vinken and
C. D. Bahler and M. Tann and C. Kesch$^*$ and T. Telli$^*$
and P. K. Chiu and K. K. Wu and F. Zattoni and L.
Evangelista and F. Ceci and M. Miszczyk and P. Rajwa and F.
Barletta and G. Gandaglia and J. A. Van Basten and M. J.
Scheltema and H. H. E. Van Melick and R. C. N. Van den Bergh
and C. A. T. Van den Berg and G. Marra and T. F. W.
Soeterik},
collaboration = {E. P. C. W. Party},
title = {{E}xternal validation of nomograms including {PSMA} {PET}
information for the prediction of lymph node involvement of
prostate cancer.},
journal = {European journal of nuclear medicine and molecular imaging},
volume = {52},
number = {10},
issn = {1619-7070},
address = {Heidelberg [u.a.]},
publisher = {Springer-Verl.},
reportid = {DKFZ-2025-00695},
pages = {3744-3756},
year = {2025},
note = {2025 Aug;52(10):3744-3756},
abstract = {Novel nomograms predicting lymph node involvement (LNI) of
prostate cancer (PCa) including PSMA PET information have
been developed. However, their predictive accuracy in
external populations is still unclear.To externally validate
four LNI nomograms including PSMA PET parameters (three
Muehlematter models and the Amsterdam-Brisbane-Sydney model)
as well as the Briganti 2012 and MSKCC nomograms.Patients
with histologically confirmed PCa undergoing preoperative
MRI and PSMA PET/CT before radical prostatectomy (RP) and
extended pelvic lymph node dissection (ePLND) were included.
Model discrimination (AUC), calibration and net benefit
using decision curve analysis were determined for each
nomogram.A total of 437 patients were included, comprising
$0.7\%$ with low-risk disease, $39.8\%$ with
intermediate-risk disease, and $59.5\%$ with high-risk
disease. Among them, 86 out of 437 $(19.7\%)$ had pN1
disease. The sensitivity and specificity of PSMA PET/CT for
the detection of LNI were $47.7\%$ $(95\%$ CI: 36.8-58.7)
and $95.4\%$ $(95\%$ CI: 92.7-97.4), respectively. Among
predictive models, the Amsterdam-Brisbane-Sydney model
achieved the highest discrimination (AUC: 0.81, $95\%$ CI:
0.76-0.86), followed by Muehlematter Model 1 (AUC: 0.79,
$95\%$ CI: 0.74-0.85), both with good calibration but slight
systematic overestimation of risks across all thresholds.
The MSKCC and Briganti 2012 models had AUCs of 0.68 $(95\%$
CI: 0.61-0.74) and 0.67 $(95\%$ CI: 0.61-0.73),
respectively, and both had moderate calibration. Decision
curve analysis indicated that the Amsterdam-Brisbane-Sydney
model provided superior net benefit across thresholds of
$5-20\%,$ followed by the Muehlematter Model 1 nomogram
showing benefit in the $14-20\%$ range. Using thresholds of
$8\%$ for the Amsterdam-Brisbane-Sydney nomogram and $15\%$
for Muehlematter Model 1, ePLND could be spared in $15\%$
and $16\%$ of patients, respectively, without missing any
LNI cases.External validation of the Muehlematter Model 1
and Amsterdam-Brisbane-Sydney nomograms for predicting LNI
confirmed their strong model discrimination, moderate
calibration, and good clinical utility, supporting their
reliability as tools to guide clinical decision-making.},
keywords = {External validation (Other) / Lymph node involvement
(Other) / Nomogram (Other) / PSMA PET/CT (Other) / Prostate
cancer (Other)},
cin = {ED01},
ddc = {610},
cid = {I:(DE-He78)ED01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40172694},
doi = {10.1007/s00259-025-07241-y},
url = {https://inrepo02.dkfz.de/record/300233},
}