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@ARTICLE{Engels:301476,
author = {P. Engels and A. Szolek and S. Hörner$^*$ and G. V.
Syrigos and K. Hebbel and M. Schmidtke and M. Zhou and M.
Mateo-Tortola and C. Schönfeld and S. Stefanczyk$^*$ and K.
Wolter$^*$ and S. Babaei and M. Schindler and M. Claassen
and D. Dauch$^*$ and L. Zender$^*$ and A. Tapía-Abellán
and A. Weber$^*$},
title = {{A}ctionable heterogeneity of hepatocellular carcinoma
therapy-induced senescence.},
journal = {Cancer immunology immunotherapy},
volume = {74},
number = {7},
issn = {0340-7004},
address = {Heidelberg},
publisher = {Springer},
reportid = {DKFZ-2025-01018},
pages = {207},
year = {2025},
abstract = {Therapy-induced senescence (TIS) is a stable cell cycle
arrest in cancerous cells favoring immune control upon
immune cell recruitment and activation via a
senescence-associated secretory phenotype (SASP). Numerous
studies have investigated the therapeutic applicability of
TIS in hepatocellular carcinoma (HCC), a frequent cancer
with high morbidity and mortality. Despite these efforts, a
comprehensive understanding of how TIS may expose
vulnerabilities specifically for immunotherapies, a potent
means of cancer therapy, in HCC remains incomplete.
Therefore, we conducted systematic studies to carefully
characterize actionable and shared SASP- or other
senescence-associated molecular parameters of TIS. We
systematically compared the TIS inducers, etoposide and
alisertib with a novel TIS inducer, CX5461, for their
effects on SASP, surfaceome and innate immune clearance of
representative human HCC cell lines. Surprisingly, all three
compounds induced both metastasis surface antigens but also
immunotherapeutically tractable antigens like CD95 (Fas),
CD276 (B7-H3) and CD340 (Her2). This was verified in four
representative HCC cell lines and publicly available
datasets of HCC. Interestingly, alisertib, etoposide and
CX5461 rendered senescent HCC vulnerable to be targeted by
either T-cell-engaging bispecific antibodies or CAR NK
cells. Collectively, our study indicates that heterogenous,
but selective features of HCC senescence may be exploited by
different immunotherapeutic approaches.},
keywords = {Humans / Carcinoma, Hepatocellular: pathology / Carcinoma,
Hepatocellular: immunology / Carcinoma, Hepatocellular: drug
therapy / Carcinoma, Hepatocellular: therapy / Liver
Neoplasms: pathology / Liver Neoplasms: immunology / Liver
Neoplasms: drug therapy / Liver Neoplasms: therapy /
Cellular Senescence: drug effects / Pyrimidines:
pharmacology / Cell Line, Tumor / Etoposide: pharmacology /
Azepines: pharmacology / Senescence-Associated Secretory
Phenotype: drug effects / Immunotherapy: methods /
Hepatocellular carcinoma (Other) / Immunology (Other) /
Immunotherapy (Other) / Senescence (Other) / Pyrimidines
(NLM Chemicals) / MLN 8237 (NLM Chemicals) / Etoposide (NLM
Chemicals) / Azepines (NLM Chemicals)},
cin = {TU01},
ddc = {610},
cid = {I:(DE-He78)TU01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40374812},
pmc = {pmc:PMC12081809},
doi = {10.1007/s00262-025-04060-w},
url = {https://inrepo02.dkfz.de/record/301476},
}