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@ARTICLE{OBrien:302327,
author = {D. O'Brien and E. Alhathli and C. Harwood and D.
Bhattacharya and K. Gupta and T. Julian and M. Weinreich$^*$
and R. J. H. West and D. Wang and R. P. Byrne and R. L.
McLaughlin and J. Wuu and M. Benatar and J. Cooper-Knock and
P. J. Shaw},
title = {{E}xtreme exercise in males is linked to m{TOR} signalling
and onset of amyotrophic lateral sclerosis.},
journal = {Brain},
volume = {nn},
issn = {0006-8950},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {DKFZ-2025-01322},
pages = {nn},
year = {2025},
note = {epub},
abstract = {Amyotrophic lateral sclerosis (ALS) is thought to be caused
by interaction between genetic and environmental factors
leading to motor neuron (MN) degeneration. Physical exercise
has been linked to ALS but controversy remains. A key
question is to determine which individuals might be at risk
of exercise-associated ALS, because unnecessary avoidance of
exercise could be harmful. We implemented complementary
strategies including Mendelian randomization and multiple
questionnaire-based measures of physical exercise in
different cohorts. We include a prospective study in UK
Biobank participants where we could test for a relationship
between exercise and the timing of future ALS symptom onset.
To interrogate the molecular basis of our observations we
performed a genetic association study of 'extreme' exercise,
equivalent to >6 hours of strenuous exercise or >12 hours of
any leisure-time exercise per week. Our data suggest that
the link between increased physical exercise and ALS is
particularly important for males who perform the most
activity; with no evidence of a link in females. We
determined that extreme exercise in males is associated with
loss-of-function genetic variants within a number of
mammalian target of rapamycin (mTOR) signalling genes that
are also differentially expressed in ALS spinal cord.
Activity-induced mTOR signalling has been shown to
selectively benefit MN. Therefore, our findings could imply
that moderate exercise is neuroprotective via enhanced mTOR
signalling, but extreme exercise in men is associated with
neurotoxicity and ALS via a failure of this mechanism. There
was no significant overlap between genes associated with
extreme exercise and those associated with ALS risk,
consistent with a true gene-environment interaction rather
than a shared genetic basis. We are not yet able to make
individual-level recommendations regarding exercise and risk
of ALS, but our conclusions should focus future
investigation.},
keywords = {Exercise (Other) / amyotrophic lateral sclerosis (ALS)
(Other) / gene-environment interaction (Other) / mammalian
target of rapamycin (mTOR) signalling (Other)},
cin = {A230},
ddc = {610},
cid = {I:(DE-He78)A230-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40577240},
doi = {10.1093/brain/awaf235},
url = {https://inrepo02.dkfz.de/record/302327},
}