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@ARTICLE{Katzengruber:302838,
author = {L. Katzengruber and P. Sander and S. Zwirner and A. Rasch
and E. Eberlein and R. Selig and W. Albrecht and L.
Zender$^*$ and S. A. Laufer$^*$},
title = {{D}iscovery of the {F}irst {H}ighly {S}elective
1,4-dihydropyrido[3,4-b]pyrazin-3(2{H})-one {MKK}4
{I}nhibitor.},
journal = {Journal of medicinal chemistry},
volume = {68},
number = {14},
issn = {0095-9065},
address = {Washington, DC},
publisher = {ACS},
reportid = {DKFZ-2025-01378},
pages = {14782-14805},
year = {2025},
note = {2025 Jul 24;68(14):14782-14805},
abstract = {Due to limited treatment options, liver failure remains a
major challenge in modern medicine. With the validation of
mitogen-activated protein kinase kinase 4 (MKK4, also known
as MEK4 or MAP2K4) as a regulator of hepatocyte
regeneration, a promising target for curative treatment of
degenerative liver diseases was recently identified via in
vivo RNAi experiments. The field of small molecules
targeting MKK4 is of growing interest. Several MKK4
inhibitors with differing scaffolds are known, but few have
reasonable selectivity profiles and drug-like properties. To
further explore the space of drug-like MKK4 scaffolds, we
performed a broad screening campaign and identified BI-D1870
as a promising candidate. The dihydropteridinone BI-D1870 is
an unselective ribosomal S6 kinase inhibitor with broad
off-target activity. In the study presented herein, we
report a successful off-to-on target strategy that led to
the development of highly selective
1,4-dihydropyrido[3,4-b]pyrazin-3(2H)-one inhibitors of
MKK4.},
cin = {TU01},
ddc = {610},
cid = {I:(DE-He78)TU01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40643363},
doi = {10.1021/acs.jmedchem.5c00919},
url = {https://inrepo02.dkfz.de/record/302838},
}