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@ARTICLE{Peng:303117,
author = {X. Peng and Ž. Janićijević and L. R. Loureiro and L.
Hoffmann and P. S. Lee and I. Cela and B. Kruppke and A.
Kegler and A. Feldmann and I. Gorodetska and A. M. Markl and
A. Dubrovska and A. K. Offermann and M. Bachmann$^*$ and L.
Baraban},
title = {{M}icrophysiological {S}olid {T}umor {M}odels in {H}ydrogel
{B}eads for {CAR} {T} {C}ell {I}mmunotherapy {E}valuation.},
journal = {Advanced science},
volume = {nn},
issn = {2198-3844},
address = {Weinheim},
publisher = {Wiley-VCH},
reportid = {DKFZ-2025-01536},
pages = {nn},
year = {2025},
note = {epub},
abstract = {Micrometastases are challenging to resect surgically and to
detect with in vivo imaging. Immunotherapy is highly
anticipated to revolutionize their treatment, but its
overall efficacy still remains limited for solid tumors.
Here, a 3D micrometastases model is developed to mimic key
microenvironmental cues, enabling in vitro evaluation of
chimeric antigen receptor (CAR) T cell immunotherapy.
Prostate cancer that preferentially metastasizes to, e.g.,
liver or bone marrow, is utilized as a model. Hydrogel beads
with an elastic modulus matching those of soft organs are
used to support long-term culturing, immunostaining, and
monitoring of the spheroids. As a biochemical cue, the
impact of fibroblast activation protein (FAP), an emerging
target in the tumor microenvironment, is investigated on
prostate cancer spheroids and on the efficacy of CAR T cell
therapy. The multi-spheroid model consists of prostate stem
cell antigen (PSCA)-expressing prostate cancer cells and
FAP-producing fibrosarcoma cells in varying ratios. The
morphological features of the model are compared to clinical
histopathology and metastatic murine model samples. Finally,
CAR T cell trials demonstrate successful chemoattraction and
infiltration through the hydrogel matrix, with a
dual-targeting approach against FAP and PSCA antigens
showing synergistic efficacy. This research provides
invaluable insights for engineering 3D tumor models and
modeling therapies targeting small metastatic or residual
tumors, suggesting that co-targeting may be a more effective
strategy to unlock the tumor microenvironment's
suppression.},
keywords = {PEGDA hydrogel beads (Other) / droplet microfluidics
(Other) / fibroblast activation protein (Other) /
immunotherapy (Other) / micrometastases (Other) / tumor
microenvironment (Other)},
cin = {DD01},
ddc = {624},
cid = {I:(DE-He78)DD01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40704837},
doi = {10.1002/advs.202508267},
url = {https://inrepo02.dkfz.de/record/303117},
}