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@ARTICLE{Liang:303228,
author = {Y. Liang$^*$ and L. Mai$^*$ and J. Schneeweiss$^*$ and R.
Lopez Perez$^*$ and M. Kirschfink and P. Huber$^*$},
title = {{R}adiotherapy {U}pregulates the {E}xpression of
{M}embrane-{B}ound {N}egative {C}omplement {R}egulator
{P}roteins on {T}umor {C}ells and {L}imits
{C}omplement-{M}ediated {T}umor {C}ell {L}ysis.},
journal = {Cancers},
volume = {17},
number = {14},
issn = {2072-6694},
address = {Basel},
publisher = {MDPI},
reportid = {DKFZ-2025-01579},
pages = {2383},
year = {2025},
note = {#EA:E055#LA:E055#},
abstract = {Background/Objectives: Radiotherapy (RT) is a mainstay of
clinical cancer therapy that causes broad immune responses.
The complement system is a pivotal effector mechanism in the
innate immune response, but the impact of RT is less well
understood. This study investigates the interaction between
RT and the complement system as a possible approach to
improve immune responses in cancer treatment. Methods: Human
solid cancer (lung, prostate, liver, breast cancer),
lymphoma, and leukemia cells were irradiated using X-rays
and treated with polyclonal antibodies or anti-CD20
monoclonal antibodies, respectively. Chromium release assay
was applied to measure cell lysis after radiation with or
without complement-activating antibody treatment. The
expression of membrane-bound complement regulatory proteins
(mCRPs; CD46, CD55, CD59), which confer resistance against
complement activation, CD20 expression, apoptosis, and
radiation-induced DNA double-strand breaks (γH2AX), was
measured by flow cytometry. The radiosensitivity of tumor
cells was assessed by colony-forming assay. Results: We
demonstrate that RT profoundly impacts complement function
by upregulating the expression of membrane-bound complement
regulatory proteins (mCRPs) on tumor cells in a dose- and
time-dependent manner. Impaired complement-mediated tumor
cell lysis could thus potentially contribute to
radiotherapeutic resistance. We also observed RT-induced
upregulation of CD20 expression on lymphoma and leukemic
cells. Notably, complement activation prior to RT proved
more effective in inducing RT-dependent early apoptosis
compared to post-irradiation treatment. While complement
modulation does not significantly alter RT-induced
DNA-damage repair mechanisms or intrinsic radiosensitivity
in cancer cells, our results suggest that combining RT with
complement-based anti-cancer therapy may enhance
complement-dependent cytotoxicity (CDC) and apoptosis in
tumor cells. Conclusions: This study sheds light on the
complex interplay between RT and the complement system,
offering insights into potential novel combinatorial
therapeutic strategies and a potential sequential structure
for certain tumor types.},
keywords = {CD20 (Other) / DNA-damage repair (Other) / complement
activation (Other) / complement-dependent cytotoxicity (CDC)
(Other) / membrane-bound complement regulatory proteins
(mCRPs) (Other) / radiosensitivity (Other) / radiotherapy
(Other) / timing (Other)},
cin = {E055},
ddc = {610},
cid = {I:(DE-He78)E055-20160331},
pnm = {315 - Bildgebung und Radioonkologie (POF4-315)},
pid = {G:(DE-HGF)POF4-315},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40723265},
doi = {10.3390/cancers17142383},
url = {https://inrepo02.dkfz.de/record/303228},
}