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@ARTICLE{Misak:304992,
author = {M. Misak and A. Basu and C. Niehrs$^*$},
title = {{S}ubfamily-selective {PCR} primers for the human {LINE}1
{L}1{PA} lineage.},
journal = {Scientific reports},
volume = {15},
number = {1},
issn = {2045-2322},
address = {[London]},
publisher = {Springer Nature},
reportid = {DKFZ-2025-01983},
pages = {32499},
year = {2025},
note = {#LA:A050# / DKFZ-ZMBH Alliance},
abstract = {Long interspersed nuclear element-1 (LINE1)
retrotransposons are classified into different subfamilies
based on evolutionary history. For human biology, the L1PA
lineage of LINE1s is particularly significant. This lineage
contains both retrotransposition-competent and inactive
subfamilies. PCR-based methods are widely used to monitor
LINE1s in genomic DNA. However, PCR analysis distinguishing
between L1PA lineage subfamilies of different evolutionary
age is thus far challenging due to the difficulty to design
primers that are sufficiently specific. Here, we developed
and applied a workflow to design PCR primers that
discriminate between different subfamilies of the L1PA
lineage. Amplicon sequencing after PCR amplification of
genomic DNA confirmed that the primers differentiate between
groups of L1PA subfamilies of different evolutionary age. We
validated the primers using RT-qPCR and verified consistency
with publicly available RNA-seq data. Moreover, inhibition
of DNA methyltransferase dose-dependently increased LINE1
expression for younger L1PA subfamilies, consistent with
transcriptional de-repression. Thus, our primers enable
PCR-based surveys that can stratify expression, copy number
or epigenetic modification for L1PA subfamilies of differing
evolutionary age.},
keywords = {Long Interspersed Nucleotide Elements: genetics / Humans /
DNA Primers: genetics / Polymerase Chain Reaction: methods /
Evolution, Molecular / L1PA (Other) / LINE1 (Other) / PCR
(Other) / Primer (Other) / Subfamily (Other) / DNA Primers
(NLM Chemicals)},
cin = {A050},
ddc = {600},
cid = {I:(DE-He78)A050-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40940398},
pmc = {pmc:PMC12432230},
doi = {10.1038/s41598-025-17649-z},
url = {https://inrepo02.dkfz.de/record/304992},
}