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@ARTICLE{Richardson:305756,
author = {T. Richardson and E. Mai and E. Menis and A. Benner$^*$ and
D. Tichy$^*$ and K. Miah$^*$ and M. Hänel and B. Besemer
and A. Boquoi and I. W. Blau and C. S. Michel and H. W.
Lindemann and S. Janjetovic and P. Brossart and H. Bernhard
and P. Reimer and H. Salwender and D. Hose and A. Seckinger
and M. Raab and H. Goldschmidt and C. Scheid},
collaboration = {G. M. M. Group},
othercontributors = {K. F. K. Studien and K. Heidelberg and F. H. U. Onkologie
and P. H. Onkologie and F. Onkologie and C. K. B.
Mergentheim and M. Baden-Baden and C. U. Berlin and C. U.
Berlin and H. K. Berlin-Buch and M. Versorgungszentrum and
F. Onkologie and O. Seestrasse and K. B. Mitte and F.
Onkologie and U. Bonn and J. K. Bonn and O. Rheinsieg and S.
K. Braunschweig and O. Schwerpunktpraxis and O. P. I. K.
Buchholz and O. Schwerpunktpraxis and K. Chemnitz and R. K.
GmbH and C.-T.-K. Cottbus and K. Darmstadt and O.
Schwerpunktpraxis and G. F. H. U. Onkologie and F. F. H. U.
Onkologie and U. Essen and E. K. E. Z. F. I. Medizin and U.
Frankfurt and A. M. Versorgungszentrum and M. Frankfurt and
K. Nordwest and I. Facharztzentrum and I. Frankfurt and V.
GmbH and F. Rotkreuz-Kliniken and P. F. I. O. U.
Hämatologie and O. Facharztpraxis and F. F. H. U. Onkologie
and K. K. Hagen and A. K. Altona and A. K. St Georg and H.
P. Altona and F. F. H. U. Onkologie and O. L. Ug and E. K.
H. gGmbH and O. Schwerpunktpraxis and K. H. GmbH and K. R.
Hannover and O. A. O. Hannover and U. Heidelberg and O.
Schwerpunktpraxis and O. S. Heilbronn and S. K. H. GmbH and
F. F. H. U. Tumorerkrankungen and U. d. Saarlandes and K.
Idar-Oberstein and S. F. H. U. Onkologie and G. F.
Hämatologie and U. Köln and O. Köln and P. I. O. U.
Hämatologie and K. Köln and F. F. Hämatologie and O. Z.
Lebach and K. d. S. L. A. Rhein and O. S. Lüneburg and U.
der Johannes Gutenberg-Universität Mainz and M.
Facharztzentrum and U. Mannheim and M. O. Praxis and F. F.
I. Medizin and P. F. I. Medizin and A. J. W. K. Minden and
K. M. Hilf and S. K. München and F. F. I. Medizin and M.
Kliniken and O. Facharztpraxis and P. Kliniken and M.
Kliniken and M. V. A. S. S. Trudbert-Klinikum and O. P.
Pinneberg and G. I. Medizin/Onkologie and F. F. Onkologie
and D. S. Hall and Z. F. A. H. U. Onkologie and D. K.
Jung-Stilling and G. O. Bodensee and O. S. Speyer and M.
Stuttgart and K. der Barmherzigen Brüder Trier and K. M.
der Borromäerinnen Trier and O. S. A. B. Trier and O.
Rheinsieg and U. Tübingen and O. O. M. GmbH and F. F.
Onkologie and A. Klinik and F. F. Onkologie},
title = {{P}rognostic {I}mpact of the {H}evylite {A}ssay in
{P}atients {W}ith {I}g{G} or {I}g{A} {M}ultiple {M}yeloma
{T}reated {W}ithin the {GMMG}-{MM}5 {T}rial.},
journal = {European journal of haematology},
volume = {nn},
issn = {0902-4441},
address = {Oxford},
publisher = {Wiley-Blackwell},
reportid = {DKFZ-2025-02376},
pages = {nn},
year = {2025},
note = {epub},
abstract = {Response assessment during treatment of multiple myeloma
(MM) typically relies on immunofixation and serum
electrophoresis. However, low levels of IgG and especially
IgA paraprotein are difficult to quantify reliably. The
Hevylite Assay quantifies the kappa and lambda fractions of
IgG and IgA separately and is useful to determine response
to therapy. Using serum samples of 360 evaluable patients
from the prospective GMMG-MM5 trial (EudraCT-No.
2010-019173-16) we assessed the normalization of the
kappa/lambda ratio with the Hevylite Assay (HLCr) at
baseline, after induction, mobilization, autologous blood
stem cell transplantation, consolidation and every three
months during maintenance or follow-up within two years
after the end of consolidation. We observed a steady
increase in the proportion of patients with normalized HLCr
over the course of therapyAchieving HLCr normalization any
time until the end of consolidation was associated with a
trend towards a prolonged progression-free survival (PFS;
hazard ratio (HR) = 0.75, $95\%$ confidence interval $(95\%$
CI) = 0.56-1.01, p = 0.06) but not overall survival (OS; HR
= 0.94, $95\%$ CI = 0.69-1.26, p = 0.66) in multivariable
time-dependent Cox regression analyses. Using a landmark
analysis from the end of consolidation there was again a
marginally statistically significant effect of HLCr
normalization by the end of consolidation on PFS using a
multivariable Cox model on the subset of the two study arms
with continuous lenalidomide maintenance (HR 0.61, $95\%$ CI
0.37-1.02, p = 0.06). No such effect was observed in study
arms in which maintenance was only applied to patients not
in CR at the end of consolidation. In conclusion, our
analysis of the Hevylite Assay in patients with IgG or IgA
myeloma from the GMMG-MM5 study did not find evidence to
support the general use of HLCr normalization as a response
parameter for predicting PFS or OS. However, the
differential effects of HLCr normalization depending on the
way in which treatment was adapted to response may be of
interest for future study designs on response-adapted
therapy. Trial Registration: ISRCTN05622749.},
keywords = {heavylite assay (Other) / multiple myeloma (Other) /
prognostic biomarker (Other)},
cin = {C060},
ddc = {610},
cid = {I:(DE-He78)C060-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41216847},
doi = {10.1111/ejh.70061},
url = {https://inrepo02.dkfz.de/record/305756},
}