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@ARTICLE{Zitricky:306672,
author = {F. Zitricky and K. Sundquist and J. Sundquist and A.
Försti$^*$ and A. Hemminki and K. Hemminki$^*$},
title = {{S}econd {P}rimary {L}ung {C}ancer {A}ssociated {W}ith
{F}amily {H}istory of {L}ung {C}ancer.},
journal = {Cancer medicine},
volume = {14},
number = {23},
issn = {2045-7634},
address = {Hoboken, NJ},
publisher = {Wiley},
reportid = {DKFZ-2025-02665},
pages = {e71431},
year = {2025},
note = {#LA:C020# Z999},
abstract = {Familial clustering of initial primary lung cancer (IPLC)
may be related to shared smoking habits, other environmental
exposures and hereditary factors, but whether familial risk
also influences the risk of second primary LC (SPLC) is not
well known. We aimed to carry out a family study between
first-degree relatives on SPLCs in Sweden.Population data on
Swedish family relationships and the diagnosed cancers were
obtained from the national registers from 1961 to 2021. IPLC
was diagnosed in 54,429 patients of whom 534 were diagnosed
with SPLC. Familial risk was assessed through the
standardized incidence ratio (SIR with $95\%$ confidence
interval) adjusted for several potential confounders,
including sex, age, calendar period, educational level and
geographic region. Familial risks were analyzed by type of
proband, histology and sex. In addition, we estimated the
effect of family history on the cumulative proportion of
patients developing SPLC by sex and histology.The estimated
SIR for SPLC was 3.98 in patients without family history and
5.24 among those with a history of lung cancer in
first-degree relatives. The SIR values depended on the
histology of IPLC and of SPLC, with the highest SIRs for
concordant histologies. For the
adenocarcinoma-adenocarcinoma sequence, SIR estimates were
5.60 and 7.51 for non-familial and familial patients,
respectively. The familial risks were further modulated by
sex and type of affected relative, with the highest SIR for
females with affected mothers (9.14).The results showed a
positive association of family history of LC with risk of
SPLC on top of high risk for SPLC in non-familial patients.
The risks differed by sex, histology and type of affected
relative. The data on family history of LC should alert
about surveillance for SPLC and may be used in future risk
stratification when eligibility for population screening is
considered.},
keywords = {Humans / Lung Neoplasms: epidemiology / Lung Neoplasms:
genetics / Lung Neoplasms: pathology / Female / Male /
Sweden: epidemiology / Middle Aged / Aged / Neoplasms,
Second Primary: epidemiology / Neoplasms, Second Primary:
genetics / Registries / Risk Factors / Incidence / Genetic
Predisposition to Disease / Adult / Aged, 80 and over /
adenocarcinoma (Other) / incidence trend (Other) / proband
(Other) / sibling risk (Other)},
cin = {B062 / HD01 / C020},
ddc = {610},
cid = {I:(DE-He78)B062-20160331 / I:(DE-He78)HD01-20160331 /
I:(DE-He78)C020-20160331},
pnm = {313 - Krebsrisikofaktoren und Prävention (POF4-313)},
pid = {G:(DE-HGF)POF4-313},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41317095},
pmc = {pmc:PMC12664089},
doi = {10.1002/cam4.71431},
url = {https://inrepo02.dkfz.de/record/306672},
}