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@ARTICLE{Gull:309653,
author = {A. Gullà and M. V. Dhodapkar and H. Einsele and M.-S.
Raab$^*$ and A. G. Solimando and C. Botta and M. Turi and L.
S. Sester and A. J. Portuguese and T. Steinbrunn and K. C.
Anderson},
title = {{R}ethinking {M}ultiple {M}yeloma {T}reatment: {T}he
{B}iological and {C}linical {I}nsights {G}uiding
{I}mmune-{B}ased {C}ombinations.},
journal = {Blood cancer discovery},
volume = {nn},
issn = {2643-3230},
address = {Philadelphia, PA},
publisher = {American Association for Cancer Research},
reportid = {DKFZ-2026-00285},
pages = {nn},
year = {2026},
note = {epub},
abstract = {The standard of care for multiple myeloma has rapidly
evolved to include immune-based therapies. However,
achieving durable immune control and long-term survival,
particularly in high-risk patients, remains difficult. In
this article, we review the immune effects of existing and
emerging therapies, dissect key drivers of resistance,
highlight rational combinations and treatment-sequencing
strategies, and summarize ongoing clinical trials that aim
to optimize durable immune control. We discuss how the
application of these biological and clinical insights may
help us rethink multiple myeloma treatment to fully
eradicate residual disease and elicit sustained natural
and/or synthetic tumor-specific immunity.Preclinical and
clinical insights are reshaping how immune-based therapies
are used in multiple myeloma. This review explores how
optimizing the integration of natural and synthetic immunity
can support a shift from disease control to deep, durable
immune eradication, paving the way for personalized immune
strategies tailored to individual immune profiles.},
cin = {A360},
ddc = {610},
cid = {I:(DE-He78)A360-20160331},
pnm = {311 - Zellbiologie und Tumorbiologie (POF4-311)},
pid = {G:(DE-HGF)POF4-311},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:41636424},
doi = {10.1158/2643-3230.BCD-25-0107},
url = {https://inrepo02.dkfz.de/record/309653},
}