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@ARTICLE{Lu:127036,
author = {K.-H. Lu$^*$ and A. Tounsi$^*$ and N. Shridhar and G.
Küblbeck$^*$ and A. Klevenz$^*$ and S. Prokosch$^*$ and T.
Bald and T. Tüting and B. Arnold$^*$},
title = {{D}ickkopf-3 {C}ontributes to the {R}egulation of
{A}nti-{T}umor {I}mmune {R}esponses by {M}esenchymal {S}tem
{C}ells.},
journal = {Frontiers in immunology},
volume = {6},
issn = {1664-3224},
address = {Lausanne},
publisher = {Frontiers Media},
reportid = {DKFZ-2017-03062},
pages = {645},
year = {2015},
abstract = {Mesenchymal stem cells (MSCs) are known to limit immune
responses in vivo by multiple soluble factors. Dickkopf-3
(DKK3), a secreted glycoprotein, has recently been
identified as a novel immune modulator. Since DKK3 has been
reported to be produced by MSCs, we investigated whether
DKK3 contributes to the immune suppression of anti-tumor
responses by MSCs. Whereas wild-type MSCs inhibited immune
responses against two different transplantation tumors,
DKK3-deficient MSCs did not affect the rejection process.
Increased CD8(+) T cell and reduced M2-type macrophages
infiltration was observed in tumors inoculated together with
DKK3-deficient MSCs. Thus, DKK3 could alter the composition
of the tumor stroma, thereby supporting the MSCs-mediated
suppression of immune responses against these tumor
transplants.},
cin = {D050},
ddc = {610},
cid = {I:(DE-He78)D050-20160331},
pnm = {314 - Tumor immunology (POF3-314)},
pid = {G:(DE-HGF)POF3-314},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:26734010},
pmc = {pmc:PMC4689786},
doi = {10.3389/fimmu.2015.00645},
url = {https://inrepo02.dkfz.de/record/127036},
}