% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Northcott:127228,
author = {P. A. Northcott$^*$ and S. Pfister$^*$ and D. Jones$^*$},
title = {{N}ext-generation (epi)genetic drivers of childhood brain
tumours and the outlook for targeted therapies.},
journal = {The lancet / Oncology},
volume = {16},
number = {6},
issn = {1470-2045},
address = {London},
publisher = {The Lancet Publ. Group},
reportid = {DKFZ-2017-03253},
pages = {e293 - e302},
year = {2015},
abstract = {Arguably, nowhere has there been a greater advance in our
understanding of biological mechanisms and potential
translational targets during the next-generation sequencing
era than in paediatric brain tumours. The so-called omics
revolution, enabled by high-throughput sequencing, has
empowered large consortia and independent groups alike to
make major genetic discoveries, from dominant-negative
histone mutations and hijacking of distal enhancer elements,
to new oncogenic gene fusions and aberrantly active gene
expression. Epigenetic deregulation has also been revealed
as a common theme across several tumour subtypes. This
Review focuses on key findings that have been transforming
the landscape of paediatric neuro-oncology research and how
these results are opening new avenues towards potential
therapeutic translation.},
keywords = {Neoplasm Proteins (NLM Chemicals)},
cin = {B062},
ddc = {610},
cid = {I:(DE-He78)B062-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:26065614},
doi = {10.1016/S1470-2045(14)71206-9},
url = {https://inrepo02.dkfz.de/record/127228},
}