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| 001 | 137690 | ||
| 005 | 20240229105115.0 | ||
| 024 | 7 | _ | |a 10.1080/23723556.2015.1074335 |2 doi |
| 024 | 7 | _ | |a pmid:30250876 |2 pmid |
| 024 | 7 | _ | |a pmc:PMC6150047 |2 pmc |
| 037 | _ | _ | |a DKFZ-2018-01567 |
| 041 | _ | _ | |a eng |
| 082 | _ | _ | |a 610 |
| 100 | 1 | _ | |a Matt, Sonja |0 P:(DE-He78)ce86d7d02a229acfaca4b63f01a1171b |b 0 |e First author |u dkfz |
| 245 | _ | _ | |a Crosstalk between p53 modifiers at PML bodies. |
| 260 | _ | _ | |a London |c 2018 |b Taylor & Francis |
| 336 | 7 | _ | |a article |2 DRIVER |
| 336 | 7 | _ | |a Output Types/Journal article |2 DataCite |
| 336 | 7 | _ | |a Journal Article |b journal |m journal |0 PUB:(DE-HGF)16 |s 1538728627_31301 |2 PUB:(DE-HGF) |
| 336 | 7 | _ | |a ARTICLE |2 BibTeX |
| 336 | 7 | _ | |a JOURNAL_ARTICLE |2 ORCID |
| 336 | 7 | _ | |a Journal Article |0 0 |2 EndNote |
| 520 | _ | _ | |a Tumor protein p53 (TP53, best known as p53), the most frequently mutated tumor suppressor in cancer, plays a central role in cell fate decisions induced by DNA damage. Regulation of p53 activity by post-translational modifications has been linked to promyelocytic leukemia nuclear bodies (PML-NBs), where p53 encounters many of its regulators. Recent evidence implies that crosstalk between p53 regulators at the PML-NB shapes post-translational modifications and function of p53. |
| 536 | _ | _ | |a 311 - Signalling pathways, cell and tumor biology (POF3-311) |0 G:(DE-HGF)POF3-311 |c POF3-311 |f POF III |x 0 |
| 588 | _ | _ | |a Dataset connected to CrossRef, PubMed, |
| 700 | 1 | _ | |a Hofmann, Thomas |0 P:(DE-He78)99ae95278bd95e30462a4fb2d12026c6 |b 1 |e Last author |u dkfz |
| 773 | _ | _ | |a 10.1080/23723556.2015.1074335 |g Vol. 5, no. 3, p. e1074335 - |0 PERI:(DE-600)2835425-4 |n 3 |p e1074335 - |t Molecular & cellular oncology |v 5 |y 2018 |x 2372-3556 |
| 909 | C | O | |o oai:inrepo02.dkfz.de:137690 |p VDB |
| 910 | 1 | _ | |a Deutsches Krebsforschungszentrum |0 I:(DE-588b)2036810-0 |k DKFZ |b 0 |6 P:(DE-He78)ce86d7d02a229acfaca4b63f01a1171b |
| 910 | 1 | _ | |a Deutsches Krebsforschungszentrum |0 I:(DE-588b)2036810-0 |k DKFZ |b 1 |6 P:(DE-He78)99ae95278bd95e30462a4fb2d12026c6 |
| 913 | 1 | _ | |a DE-HGF |l Krebsforschung |1 G:(DE-HGF)POF3-310 |0 G:(DE-HGF)POF3-311 |2 G:(DE-HGF)POF3-300 |v Signalling pathways, cell and tumor biology |x 0 |4 G:(DE-HGF)POF |3 G:(DE-HGF)POF3 |b Gesundheit |
| 914 | 1 | _ | |y 2018 |
| 915 | _ | _ | |a DBCoverage |0 StatID:(DE-HGF)0300 |2 StatID |b Medline |
| 920 | 1 | _ | |0 I:(DE-He78)A210-20160331 |k A210 |l Zelluläre Seneszenz |x 0 |
| 980 | _ | _ | |a journal |
| 980 | _ | _ | |a VDB |
| 980 | _ | _ | |a I:(DE-He78)A210-20160331 |
| 980 | _ | _ | |a UNRESTRICTED |
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