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@ARTICLE{Erhardt:153080,
      author       = {S. Erhardt and G. Stoecklin},
      title        = {{T}he heat's on: nuclear stress bodies signal intron
                      retention.},
      journal      = {The EMBO journal},
      volume       = {39},
      number       = {3},
      issn         = {1460-2075},
      address      = {Hoboken, NJ [u.a.]},
      publisher    = {Wiley},
      reportid     = {DKFZ-2020-00162},
      pages        = {e104154},
      year         = {2020},
      note         = {DKFZ-ZMBH Alliance2020 Feb 3;39(3):e104154},
      abstract     = {The cellular response to heat shock requires massive
                      adaptation of gene expression driven by the transcription
                      factor HSF1, which assembles in nuclear stress bodies
                      together with human satellite III RNA and numerous splicing
                      factors. In this issue of The EMBO Journal, Ninomiya et al
                      demonstrate that nuclear stress bodies serve as a platform
                      for phosphorylation of the SR protein SRSF9 by the CLK1
                      kinase, which promotes retention of a large number of
                      introns during the recovery phase from heat shock.},
      subtyp        = {Editorial},
      cin          = {A200},
      ddc          = {570},
      cid          = {I:(DE-He78)A200-20160331},
      pnm          = {311 - Signalling pathways, cell and tumor biology
                      (POF3-311)},
      pid          = {G:(DE-HGF)POF3-311},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:31919860},
      doi          = {10.15252/embj.2019104154},
      url          = {https://inrepo02.dkfz.de/record/153080},
}