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@ARTICLE{Pedersen:153184,
author = {M. S. Pedersen and M. Müller and T. Rülicke and N.
Leitner and R. Kain and H. Regele and S. Wang$^*$ and H.-J.
Gröne$^*$ and S. Rong and H. Haller and F. Gueler and A. J.
Rees and D. Kerjaschki},
title = {{L}ymphangiogenesis in a mouse model of renal transplant
rejection extends life span of the recipients.},
journal = {Kidney international},
volume = {97},
number = {1},
issn = {0085-2538},
address = {New York, NY},
publisher = {Elsevier},
reportid = {DKFZ-2020-00237},
pages = {89 - 94},
year = {2020},
abstract = {Renal allograft rejection can be prevented by immunological
tolerance, which may be associated with de novo formed
lymphatic vessels in the donor kidney after transplantation
in man. A suitable mouse model of renal allograft rejection
in which lymphangiogenesis can be deliberately induced in
the graft is critical for elucidating the mechanisms
responsible for the association between attenuated
transplant rejection and abundance of lymphatic vessels.
Here we describe the development of a novel mouse model of
rapid renal transplant rejection in which transgenic
induction of lymphangiogenesis in the immune-incompatible
graft greatly extends its survival time. Thus, our novel
approach may facilitate exploitation of lymphangiogenesis in
the grafted organ.},
cin = {G130},
ddc = {610},
cid = {I:(DE-He78)G130-20160331},
pnm = {319H - Addenda (POF3-319H)},
pid = {G:(DE-HGF)POF3-319H},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:31718844},
doi = {10.1016/j.kint.2019.07.027},
url = {https://inrepo02.dkfz.de/record/153184},
}