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@ARTICLE{Packer:153719,
author = {R. J. Packer and A. Iavarone and D. T. W. Jones$^*$ and J.
O. Blakeley and E. Bouffet and M. J. Fisher and E. Hwang and
C. Hawkins and L. Kilburn and T. MacDonald and S. M.
Pfister$^*$ and B. Rood and F. J. Rodriguez and U. Tabori
and V. Ramaswamy and Y. Zhu and J. Fangusaro and S. A.
Johnston and D. H. Gutmann},
title = {{I}mplications of {N}ew {U}nderstandings of {G}liomas in
{C}hildren and {A}dults with {NF}1: {R}eport of a
{C}onsensus {C}onference.},
journal = {Neuro-Oncology},
volume = {22},
number = {6},
issn = {1523-5866},
address = {Oxford},
publisher = {Oxford Univ. Press},
reportid = {DKFZ-2020-00416},
pages = {773-784},
year = {2020},
note = {2020 Jun 9;22(6):773-784},
abstract = {Gliomas are the most common primary central nervous system
tumors occurring in children and adults with
neurofibromatosis type I (NF1). Over the past decade,
discoveries of the molecular basis of low-grade gliomas
(LGGs) have led to new approaches for diagnosis and
treatments. However, these new understandings have not been
fully applied to the management of NF1-associated gliomas. A
consensus panel consisting of experts in NF1 and gliomas was
convened to review the current molecular knowledge of
NF1-associated low grade 'transformed' and high-grade
gliomas; insights gained from mouse models of NF1-LGGs;
challenges in diagnosing and treating older patients with
NF1-associated gliomas; and advances in molecular targeted
treatment and potential immunologic treatment of these
tumors. Next steps are recommended to advance the management
and outcomes for NF1-associated gliomas.},
cin = {B360 / B062},
ddc = {610},
cid = {I:(DE-He78)B360-20160331 / I:(DE-He78)B062-20160331},
pnm = {312 - Functional and structural genomics (POF3-312)},
pid = {G:(DE-HGF)POF3-312},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:32055852},
doi = {10.1093/neuonc/noaa036},
url = {https://inrepo02.dkfz.de/record/153719},
}