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@ARTICLE{Moore:153956,
      author       = {A. Moore and E. Kane and Z. Wang and O. A. Panagiotou and
                      L. R. Teras and A. Monnereau and N. Wong Doo and M. J.
                      Machiela and C. F. Skibola and S. L. Slager and G. Salles
                      and N. J. Camp and P. M. Bracci and A. Nieters and R. C. H.
                      Vermeulen and J. Vijai and K. E. Smedby and Y. Zhang and C.
                      M. Vajdic and W. Cozen and J. J. Spinelli and H. Hjalgrim
                      and G. G. Giles and B. K. Link and J. Clavel and A. A.
                      Arslan and M. P. Purdue and L. F. Tinker and D. Albanes and
                      G. M. Ferri and T. M. Habermann and H.-O. Adami and N.
                      Becker$^*$ and Y. Benavente and S. Bisanzi and P. Boffetta
                      and P. Brennan and A. R. Brooks-Wilson and F. Canzian$^*$
                      and L. Conde and D. G. Cox and K. Curtin and L. Foretova and
                      S. M. Gapstur and H. Ghesquières and M. Glenn and B.
                      Glimelius and R. D. Jackson and Q. Lan and M. Liebow and M.
                      Maynadie and J. McKay and M. Melbye and L. Miligi and R. L.
                      Milne and T. J. Molina and L. M. Morton and K. E. North and
                      K. Offit and M. Padoan and A. V. Patel and S. Piro and V.
                      Ravichandran and E. Riboli and S. de Sanjose and R. K.
                      Severson and M. C. Southey and A. Staines and C. Stewart and
                      R. C. Travis and E. Weiderpass and S. Weinstein and T. Zheng
                      and S. J. Chanock and N. Chatterjee and N. Rothman and B. M.
                      Birmann and J. R. Cerhan and S. I. Berndt},
      title        = {{G}enetically {D}etermined {H}eight and {R}isk of
                      {N}on-hodgkin {L}ymphoma.},
      journal      = {Frontiers in oncology},
      volume       = {9},
      issn         = {2234-943X},
      address      = {Lausanne},
      publisher    = {Frontiers Media},
      reportid     = {DKFZ-2020-00546},
      pages        = {1539},
      year         = {2020},
      abstract     = {Although the evidence is not consistent, epidemiologic
                      studies have suggested that taller adult height may be
                      associated with an increased risk of some non-Hodgkin
                      lymphoma (NHL) subtypes. Height is largely determined by
                      genetic factors, but how these genetic factors may
                      contribute to NHL risk is unknown. We investigated the
                      relationship between genetic determinants of height and NHL
                      risk using data from eight genome-wide association studies
                      (GWAS) comprising 10,629 NHL cases, including 3,857 diffuse
                      large B-cell lymphoma (DLBCL), 2,847 follicular lymphoma
                      (FL), 3,100 chronic lymphocytic leukemia (CLL), and 825
                      marginal zone lymphoma (MZL) cases, and 9,505 controls of
                      European ancestry. We evaluated genetically predicted height
                      by constructing polygenic risk scores using 833
                      height-associated SNPs. We used logistic regression to
                      estimate odds ratios (OR) and $95\%$ confidence intervals
                      (CI) for association between genetically determined height
                      and the risk of four NHL subtypes in each GWAS and then used
                      fixed-effect meta-analysis to combine subtype results across
                      studies. We found suggestive evidence between taller
                      genetically determined height and increased CLL risk (OR =
                      1.08, $95\%$ CI = 1.00-1.17, p = 0.049), which was slightly
                      stronger among women (OR = 1.15, $95\%$ CI: 1.01-1.31, p =
                      0.036). No significant associations were observed with
                      DLBCL, FL, or MZL. Our findings suggest that there may be
                      some shared genetic factors between CLL and height, but
                      other endogenous or environmental factors may underlie
                      reported epidemiologic height associations with other
                      subtypes.},
      cin          = {C020 / C055},
      ddc          = {610},
      cid          = {I:(DE-He78)C020-20160331 / I:(DE-He78)C055-20160331},
      pnm          = {313 - Cancer risk factors and prevention (POF3-313)},
      pid          = {G:(DE-HGF)POF3-313},
      typ          = {PUB:(DE-HGF)16},
      pubmed       = {pmid:32064237},
      pmc          = {pmc:PMC6999122},
      doi          = {10.3389/fonc.2019.01539},
      url          = {https://inrepo02.dkfz.de/record/153956},
}