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@ARTICLE{Boutin:169806,
author = {S. Boutin and D. Hildebrand and S. Boulant$^*$ and M.
Kreuter and J. Rüter and S. R. Pallerla and T. P. Velavan
and D. Nurjadi},
title = {{H}ost factors facilitating {SARS}-{C}o{V}-2 virus
infection and replication in the lungs.},
journal = {Cellular and molecular life sciences},
volume = {78},
number = {16},
issn = {1420-9071},
address = {Cham (ZG)},
publisher = {Springer International Publishing AG},
reportid = {DKFZ-2021-01560},
pages = {5953-5976},
year = {2021},
note = {2021 Aug;78(16):5953-5976},
abstract = {SARS-CoV-2 is the virus causing the major pandemic facing
the world today. Although, SARS-CoV-2 primarily causes lung
infection, a variety of symptoms have proven a systemic
impact on the body. SARS-CoV-2 has spread in the community
quickly infecting humans from all age, ethnicities and
gender. However, fatal outcomes have been linked to specific
host factors and co-morbidities such as age, hypertension,
immuno-deficiencies, chronic lung diseases or metabolic
disorders. A major shift in the microbiome of patients
suffering of the coronavirus disease 2019 (COVID-19) have
also been observed and is linked to a worst outcome of the
disease. As many co-morbidities are already known to be
associated with a dysbiosis of the microbiome such as
hypertension, diabetes and metabolic disorders. Host factors
and microbiome changes are believed to be involved as a
network in the acquisition of the infection and the
development of the diseases. We will review in detail in
this manuscript, the immune response toward SARS-CoV-2
infection as well as the host factors involved in the
facilitation and worsening of the infection. We will also
address the impact of COVID-19 on the host's microbiome and
secondary infection which also worsen the disease.},
subtyp = {Review Article},
keywords = {COVID-19 (Other) / Co-morbidities (Other) / Host factor
(Other) / Innate immune response (Other) / Microbiome
(Other) / SARS-CoV-2 (Other)},
cin = {F140},
ddc = {610},
cid = {I:(DE-He78)F140-20160331},
pnm = {316 - Infektionen, Entzündung und Krebs (POF4-316)},
pid = {G:(DE-HGF)POF4-316},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:34223911},
pmc = {pmc:PMC8256233},
doi = {10.1007/s00018-021-03889-5},
url = {https://inrepo02.dkfz.de/record/169806},
}