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@MASTERSTHESIS{Wllner:177454,
author = {A. Wüllner$^*$},
title = {{T}oxicity studies on lung and pancreatic cancer celllines:
{A}n approach towards {FLASH} mechanisms},
school = {Universität Heidelberg},
type = {Bachelorarbeit},
reportid = {DKFZ-2021-02546},
year = {2021},
note = {Corresponding author J. Seco; Bachelorarbeit, Universität
Heidelberg, 2021},
abstract = {Several studies have hinted towards involvement of radical
scavengers in the sparingof healthy tissue in radiotherapy
(RT) for ultra-high dose rates (FLASH effect). Thesaturation
of the intracellular enzyme Superoxide Dismutase (SOD) is
proposed to playa key role in the FLASH effect. The aim of
the current study was to establish treatmentprotocols to
achieve $80\%$ cell viability of two cancer cell lines for
four compounds thataffect the enzyme and its substrate
Superoxide. Cytotoxicity assays were done on theH460 lung
cancer cell line and the Panc-1 pancreatic cancer cell line
with the compoundsH2O2, MSB, NaCN and KCN. The effect of
Catalase on cell viability was tested and theeffect of NaCN
treatment in combination with conventional RT and FLASH RT
wastested using clonogenic assays on the H460 cell line.
Treatment protocols to $achieve80\%$ cell viability were
found for all four compounds and cell lines. For almost all
celllines a treatment protocol to achieve $50\%$ viability
was found. A significant increase ofviability of about
$50\%$ was detected for combining NaCN treatment with FLASH
RT.Further studies on the effects of the compounds in
combination with RT have to be doneas well as on their
effect on the SOD levels.},
cin = {E041},
cid = {I:(DE-He78)E041-20160331},
pnm = {315 - Bildgebung und Radioonkologie (POF4-315)},
pid = {G:(DE-HGF)POF4-315},
typ = {PUB:(DE-HGF)2},
url = {https://inrepo02.dkfz.de/record/177454},
}