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@ARTICLE{Liu:267555,
author = {Y. Liu and A. Keib and B. Neuber and L. Wang and A.
Riemer$^*$ and M. Bonsack$^*$ and A. Hückelhoven-Krauss and
A. Schmitt and C. Müller-Tidow and M. Schmitt},
title = {{D}efinition and {C}haracterization of {SOX}11-{D}erived
{T} {C}ell {E}pitopes towards {I}mmunotherapy of {G}lioma.},
journal = {International journal of molecular sciences},
volume = {24},
number = {3},
issn = {1422-0067},
address = {Basel},
publisher = {Molecular Diversity Preservation International},
reportid = {DKFZ-2023-00333},
pages = {1943},
year = {2023},
abstract = {The transcription factor SOX11 is a tumor-associated
antigen with low expression in normal cells, but
overexpression in glioblastoma (GBM). So far, conventional
surgery, chemotherapy, and radiotherapy have not
substantially improved the dismal prognosis of
relapsed/refractory GBM patients. Immunotherapy is
considered a promising strategy against GBM, but there is a
fervent need for better immunotargets in GBM. To this end,
we performed an in silico prediction study on SOX11, which
primarily yielded ten promising HLA-A*0201-restricted
peptides derived from SOX11. We defined a novel peptide
FMACSPVAL, which had the highest score according to in
silico prediction (6.02 nM by NetMHC-4.0) and showed an
exquisite binding affinity to the HLA-A*0201 molecule in the
peptide-binding assays. In the IFN-γ ELISPOT assays,
FMACSPVAL demonstrated a high efficiency for generating
SOX11-specific CD8+ T cells. Nine out of thirty-two healthy
donors showed a positive response to SOX11, as assessed by
the ELISPOT assays. Therefore, this novel antigen peptide
epitope seems to be promising as a target for T cell-based
immunotherapy in GBM. The adoptive transfer of in vitro
elicited SOX11-specific CD8+ T cells constitutes a potential
approach for the treatment of GBM patients.},
keywords = {HLA-A*0201 (Other) / SOX11 (Other) / glioma (Other) /
immunotherapy (Other) / tumor-associated antigen (Other)},
cin = {F130},
ddc = {540},
cid = {I:(DE-He78)F130-20160331},
pnm = {316 - Infektionen, Entzündung und Krebs (POF4-316)},
pid = {G:(DE-HGF)POF4-316},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:36768267},
doi = {10.3390/ijms24031943},
url = {https://inrepo02.dkfz.de/record/267555},
}