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000277770 1001_ $$0P:(DE-He78)1f71ccf4137962d5dbf12200b9ffa75d$$aAmaya Ramirez, Cinthia Claudia$$b0$$eFirst author$$udkfz
000277770 245__ $$aBreast Cancer Metastatic Progression Requires mRNA Posttranscriptional Suppression.
000277770 260__ $$aPhiladelphia, Pa.$$bAACR$$c2023
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000277770 500__ $$a#EA:B250#LA:B250# / 2023 Aug 1;83(15):2448-2449 / IN THE SPOTLIGHT
000277770 520__ $$aCancer cell survival is highly dependent on its metabolic reprogramming, which supports not only cell growth but also confers to the tumor cells characteristics to initiate migration and colonization. Among the different mechanisms that are involved, translational control plays a significant role in oncogenesis; however, its impact on cancer progression still remains poorly understood. A study by Navickas and colleagues revealed that the RNA-binding protein heterogeneous nuclear ribonucleoprotein C (HNRNPC) functions as a translational regulator, and its downregulation in highly metastatic cells leads to the lengthening of 3' untranslated regions in HNRNPC-bound mRNAs, resulting in translational repression mediated by the AGO-miRNA RNA-induced silencing complex.
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000277770 7001_ $$0P:(DE-He78)4af0380446b05b5f1995502016151a1b$$aLoayza-Puch, Fabricio$$b1$$eLast author$$udkfz
000277770 773__ $$0PERI:(DE-600)2036785-5$$a10.1158/0008-5472.CAN-23-1729$$gp. OF1 - OF2$$n15$$p2448-2449$$tCancer research$$v83$$x0099-7013$$y2023
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