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@ARTICLE{Hensel:302861,
author = {A. Hensel and R. Váraljai$^*$ and S. K. Knauer},
title = {{R}aising the iron curtain: {L}actate's secret role in
oxidative stress defense.},
journal = {Redox Biology},
volume = {85},
issn = {2213-2317},
address = {Amsterdam [u.a.]},
publisher = {Elsevier},
reportid = {DKFZ-2025-01401},
pages = {103754},
year = {2025},
abstract = {The hypothesis presented here is that certain cell types
under oxidative stress, such as cancer cells, reprogram
their metabolism to accumulate lactate, along with cytosolic
Fe2+ within the labile iron pool, thereby establishing a
metabolite-based H2O2 detoxification system. In this
scenario, the Fenton reaction between Fe2+ and H2O2
generates hydroxyl radicals (HO•), which are subsequently
scavenged by abundant lactate. Thus, lactate production may
function as a protective, iron-dependent antioxidant
mechanism, enabling cells to decompose H2O2 and prevent
damage to crucial biomolecules. If this system is
compromised, for instance by inadequate HO•-scavenging or
impaired Fe2+ recycling, cells may become prone to
ferroptosis.},
subtyp = {Review Article},
keywords = {Fenton reaction (Other) / Ferroptosis (Other) / Labile iron
pool (Other) / Lactate (Other) / Oxidative stress (Other)},
cin = {ED01},
ddc = {570},
cid = {I:(DE-He78)ED01-20160331},
pnm = {899 - ohne Topic (POF4-899)},
pid = {G:(DE-HGF)POF4-899},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:40663910},
doi = {10.1016/j.redox.2025.103754},
url = {https://inrepo02.dkfz.de/record/302861},
}