Journal Article

DKFZ-2025-02936

http://join2-wiki.gsi.de/foswiki/pub/Main/Artwork/join2_logo100x88.png Targeting ATR offers multifaceted treatment strategies involving RAD51-mediated compensatory DNA repair in bladder cancer.

Pannhausen, J. ; Chughtai, A. A. ; Yüce, C.-L. ; Melzer, M. K. ; Ma, Y. ; Seillier, L. ; van der Vorst, E. P. C. ; Andrieux, G. ; Wirtz, J. ; Leypold, S. ; Kühnel, M. P. ; Hoffmann, P. ; Heilmann-Heimbach, S. ; Boerries, M.DKFZ* ; Kleger, A. ; Saar, M. ; Eble, M. J. ; Jonigk, D. D. ; Gaisa, N. T. ; Rose, M.

2026
Springer Heidelberg

Keyword(s): ATR ; B02 ; Bladder cancer ; Cancer drug resistance ; Ceralasertib ; DNA damage response ; Homologous recombination repair ; RAD51 ; Squamous cell carcinoma

Note: Volume 45, article number 17, (2026)

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Contributing Institute(s):

1. DKTK Koordinierungsstelle Freiburg (FR01)

Research Program(s):

1. 899 - ohne Topic (POF4-899) (POF4-899)

Appears in the scientific report 2025

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Database coverage:
; ; Article Processing Charges ; Clarivate Analytics Master Journal List ; Current Contents - Clinical Medicine ; DOAJ Seal ; Ebsco Academic Search ; Essential Science Indicators ; Fees ; SCOPUS ; Science Citation Index Expanded ; Web of Science Core Collection

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